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      The placentas of patients with severe acute respiratory syndrome: a pathophysiological evaluation.

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          Abstract

          The pathology of the placentas delivered from pregnant women who had severe acute respiratory syndrome (SARS) in Hong Kong was studied.

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          A case‐controlled study comparing clinical course and outcomes of pregnant and non‐pregnant women with severe acute respiratory syndrome

          Chui Miu Lam, Shell Wong, Tse Leung … (2004)
          Objective  To compare the clinical courses and outcomes of pregnant severe acute respiratory syndrome (SARS) patients and non‐pregnant SARS patients. Design  A case–control study. Setting  Tertiary Hospital for Infectious Disease. Sample  Ten pregnant and 40 non‐pregnant female patients infected with SARS. Methods  Clinical course and outcomes of pregnant SARS patients were compared with a group of non‐pregnant SARS patient. Cases and controls were matched with respect to sex, age, timing of contracting SARS, health care workers status and underlying illness. Main outcome measures  The incidence of intensive care unit admission, intubation, medical complications and death rate. Results  Pregnancy had no discernible impact on clinical symptoms and presentation delay. Four out of the 10 pregnant patients, nevertheless, required endotracheal intubation and six were admitted to the intensive care unit (ICU), as compared with 12.5% intubation rate (P= 0.065) and 17.5% ICU admission rate (P= 0.012) in the non‐pregnant group. More pregnant SARS patients developed renal failure (P= 0.006) and disseminated intravascular coagulopathy (P= 0.006), as compared with non‐pregnant SARS group. There were three deaths in the pregnant group, whereas there was no death in the non‐pregnant control group (P= 0.006). Conclusion  Pregnant women with SARS experience a worse clinical course and poorer outcomes compared with non‐pregnant women.
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            Infants born to mothers with severe acute respiratory syndrome.

            T Fok, Dean F Wong, K. L. Hon … (2003)
            Severe acute respiratory syndrome (SARS) is a newly discovered infectious disease caused by a novel coronavirus. During the community outbreak in Hong Kong, 5 liveborn infants were born to pregnant women with SARS. A systematic search for perinatal transmission of the SARS-associated coronavirus, including serial reverse transcriptase-polymerase chain reaction assays, viral cultures, and paired serologic titers, failed to detect the virus in any of the infants. In addition, none of the infants developed clinical, radiologic, hematologic, or biochemical evidence suggestive of SARS. One preterm infant developed jejunal perforation and another developed necrotizing enterocolitis with ileal perforation shortly after birth. This case series is the first report to describe the clinical course of the first cohort of liveborn infants born to pregnant women with SARS.
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              SARS and Pregnancy: A Case Report

              Corwin A. Robertson, Sara Lowther, Thomas Birch … (2004)
              We report a laboratory-confirmed case of severe acute respiratory syndrome (SARS) in a pregnant woman. Although the patient had respiratory failure, a healthy infant was subsequently delivered, and the mother is now well. There was no evidence of viral shedding at delivery. Antibodies to SARS virus were detected in cord blood and breast milk.
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                Author and article information

                Journal
                Journal ID (iso-abbrev): Pathology
                Title: Pathology
                Publisher: Informa UK Limited
                ISSN: 0031-3025
                ISSN (Print): 0031-3025
                Publication date (Electronic): Jun 2006
                Volume: 38
                Issue: 3
                Affiliations
                [1 ] Department of Pathology, Princess Margaret Hospital, Lai Chi Kok, Kowloon, Hong Kong Special Administrative Region, China. ngwaifu@cuhk.edu.hk
                Article
                Publisher Item ID: JL4622VXR8453061
                DOI: 10.1080/00313020600696280
                PubMed ID: 16753741
                SO-VID: 9ecf934d-c90d-4ad4-8a73-79386396439c
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