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      Lipoprotein (a) and Low-density lipoprotein apolipoprotein B metabolism following apheresis in patients with elevated lipoprotein(a) and coronary artery disease.

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          Abstract

          Lipoprotein apheresis effectively lowers lipoprotein(a) [Lp(a)] and low-density lipoprotein (LDL) by approximately 60%-70%. The rebound of LDL and Lp(a) particle concentrations following lipoprotein apheresis allows the determination of fractional catabolic rate (FCR) and hence production rate (PR) during non-steady state conditions. We aimed to investigate the kinetics of Lp(a) and LDL apolipoprotein B-100 (apoB) particles in patients with elevated Lp(a) and coronary artery disease undergoing regular apheresis.

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          Author and article information

          Journal
          Eur. J. Clin. Invest.
          European journal of clinical investigation
          Wiley
          1365-2362
          0014-2972
          Feb 2019
          : 49
          : 2
          Affiliations
          [1 ] School of Biomedical Science, University of Western Australia, Western Australia, Australia.
          [2 ] School of Medicine, University of Western Australia, Western Australia, Australia.
          [3 ] Department of Internal Medicine IV, Ludwig-Maximilans-University Munich, Munich, Germany.
          [4 ] Department of Cardiology, Lipid Disorders Clinic, Royal Perth Hospital, Western Australia, Australia.
          Article
          10.1111/eci.13053
          30447089
          02f82d75-2b31-422c-9675-bbbd1c2d930f
          History

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