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      Erratum: Global reemergence of enterovirus D68 as an important pathogen for acute respiratory infections

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      Reviews in Medical Virology
      John Wiley and Sons Inc.

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          Abstract

          In the originally published version of Imamura and Oshitani (2015), issue 25:2, from page 102–114 of Reviews in Medical Virology 1, the authors would like to change the sentence on page 105, first column, first paragraph, line 17. The correct statement should be “In contrast, lineage 3 strains had deletions only at positions 624–704. The lineage 2 strains were not analyzed for those deletions in this study, due to the lack of sequence data of lineage 2 strains.” In Table 1, the row for “USA (Arizona) [7]” is duplicated. We would like to delete one of them. We apologize for any misunderstanding this may have caused. Published online 3 December 2014 in Wiley Online Library (wileyonlinelibrary.com) DOI: 10.1002/rmv.1820

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          Global reemergence of enterovirus D68 as an important pathogen for acute respiratory infections

          We previously detected enterovirus D68 (EV-D68) in children with severe acute respiratory infections in the Philippines in 2008–2009. Since then, the detection frequency of EV-D68 has increased in different parts of the world, and EV-D68 is now recognized as a reemerging pathogen. However, the epidemiological profile and clinical significance of EV-D68 is yet to be defined, and the virological characteristics of EV-D68 are not fully understood. Recent studies have revealed that EV-D68 is detected among patients with acute respiratory infections of differing severities ranging from mild upper respiratory tract infections to severe pneumonia including fatal cases in pediatric and adult patients. In some study sites, the EV-D68 detection rate was higher among patients with lower respiratory tract infections than among those with upper respiratory tract infections, suggesting that EV-D68 infections are more likely to be associated with severe respiratory illnesses. EV-D68 strains circulating in recent years have been divided into three distinct genetic lineages with different antigenicity. However, the association between genetic differences and disease severity, as well as the occurrence of large-scale outbreaks, remains elusive. Previous studies have revealed that EV-D68 is acid sensitive and has an optimal growth temperature of 33 °C. EV-D68 binds to α2,6-linked sialic acids; hence, it is assumed that it has an affinity for the upper respiratory track where these glycans are present. However, the lack of suitable animal model constrains comprehensive understanding of the pathogenesis of EV-D68. © 2014 The Authors. Reviews in Medical Virology published by John Wiley & Sons Ltd.
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            Author and article information

            Journal
            Rev Med Virol
            Rev. Med. Virol
            10.1002/(ISSN)1099-1654
            RMV
            Reviews in Medical Virology
            John Wiley and Sons Inc. (Hoboken )
            1052-9276
            1099-1654
            06 July 2015
            July 2015
            : 25
            : 4 ( doiID: 10.1002/rmv.v25.4 )
            : 268
            Article
            RMV1844
            10.1002/rmv.1844
            4738597
            26151890
            04bae739-dd9c-43b4-97cb-c773d06be8b2
            Copyright © 2015 John Wiley & Sons, Ltd.
            History
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            Pages: 1
            Categories
            Erratum
            Erratum
            Custom metadata
            2.0
            rmv1844
            July 2015
            Converter:WILEY_ML3GV2_TO_NLMPMC version:4.7.5 mode:remove_FC converted:29.01.2016

            Microbiology & Virology
            Microbiology & Virology

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