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Poly(ADP‐Ribose) Polymerase Inhibition Improves Erectile Function by Activation of Nitric Oxide/Cyclic Guanosine Monophosphate Pathway in Diabetic Rats
May 2012
May 2012
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Abstract
Endothelial dysfunction-induced abnormalities of the nitric oxide (NO)/cyclic guanosine
monophosphate (cGMP) signaling pathway in the corpus cavernosum are thought to be
the main factors involved in the pathogenesis of diabetes-induced erectile dysfunction
(ED). Recent studies have shown that the poly(adenosine diphosphate ribose) polymerase
(PARP) pathway plays a critical role in diabetic endothelial dysfunction.