Acute ST-segment elevation myocardial infarction is associated with decreased human antimicrobial peptide LL-37 and increased human neutrophil peptide-1 to 3 in plasma.

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Abstract

Increasing evidence indicates that antimicrobial peptides, human neutrophil peptide-1, -2, and -3 (HNP1-3) and human antimicrobial peptide LL-37 are involved in the pathophysiology of atherosclerosis; however, little is known about their circulating protein levels in acute myocardial infarction (AMI). We therefore investigated whether their plasma levels are associated with stable coronary artery disease (CAD) and acute ST-segment elevation myocardial infarction (STEMI).

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Journal

Journal ID (iso-abbrev): J. Atheroscler. Thromb.

Title: Journal of atherosclerosis and thrombosis

ISSN (Electronic): 1880-3873

ISSN (Print): 1340-3478

Publication date (Electronic): 2012

Volume: 19

Issue: 4

Affiliations

[1 ] State Key Laboratory of Cadiovascular Disease, Fuwai Hospital, National Center for Cardiovascular Diseases, Chinese Academy of Medical Sciences and Peking Union Medical College, No. 167 Beilishi Road, Beijing, China.

Article

Publisher Item ID: JST.JSTAGE/jat/10108

DOI: 10.5551/jat.10108

PubMed ID: 22186100

SO-VID: 06964c99-9de8-4949-b95f-844a1a23eb06

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