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      The Corporate Psychopaths Theory of the Global Financial Crisis

      Journal of Business Ethics
      Springer Nature

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          Limbic abnormalities in affective processing by criminal psychopaths as revealed by functional magnetic resonance imaging

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            The development of psychopathy.

            The current review focuses on the construct of psychopathy, conceptualized as a clinical entity that is fundamentally distinct from a heterogeneous collection of syndromes encompassed by the term 'conduct disorder'. We will provide an account of the development of psychopathy at multiple levels: ultimate causal (the genetic or social primary cause), molecular, neural, cognitive and behavioral. The following main claims will be made: (1) that there is a stronger genetic as opposed to social ultimate cause to this disorder. The types of social causes proposed (e.g., childhood sexual/physical abuse) should elevate emotional responsiveness, not lead to the specific form of reduced responsiveness seen in psychopathy; (2) The genetic influence leads to the emotional dysfunction that is the core of psychopathy; (3) The genetic influence at the molecular level remains unknown. However, it appears to impact the functional integrity of the amygdala and orbital/ventrolateral frontal cortex (and possibly additional systems); (4) Disruption within these two neural systems leads to impairment in the ability to form stimulus-reinforcement associations and to alter stimulus-response associations as a function of contingency change. These impairments disrupt the impact of standard socialization techniques and increase the risk for frustration-induced reactive aggression respectively.
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              When Psychopaths go to Work: A Case Study of an Industrial Psychopath

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                Author and article information

                Journal
                Journal of Business Ethics
                J Bus Ethics
                Springer Nature
                0167-4544
                1573-0697
                August 2011
                February 2011
                : 102
                : 2
                : 255-259
                Article
                10.1007/s10551-011-0810-4
                0921199a-5e39-4eec-8c96-6b1143d81743
                © 2011
                History

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