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Immunology: TL1A in the inflammatory network in autoimmune diseases.

Publication date: 2010-01-31

Keywords: Animals, Arthritis, Rheumatoid, etiology, metabolism, Humans, Receptors, Tumor Necrosis Factor, Member 25, Tumor Necrosis Factor Ligand Superfamily Member 15

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Abstract

Several lines of evidence both from experimental models and from clinical studies reveal that TNF-like ligand 1A (TL1A) and its interaction with death receptor 3 (DR3) is critically involved in the pathogenesis of rheumatoid arthritis and other autoimmune diseases.

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PubMed ID:: 20125169

DOI:: 10.1038/nrrheum.2009.263

ScienceOpen disciplines: Chemistry

Keywords: Animals,Arthritis, Rheumatoid,etiology,metabolism,Humans,Receptors, Tumor Necrosis Factor, Member 25,Tumor Necrosis Factor Ligand Superfamily Member 15

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ScienceOpen disciplines: Chemistry

Keywords: Animals, Arthritis, Rheumatoid, etiology, metabolism, Humans, Receptors, Tumor Necrosis Factor, Member 25, Tumor Necrosis Factor Ligand Superfamily Member 15

Immunology: TL1A in the inflammatory network in autoimmune diseases.

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