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      Site-specific interplay between O-GlcNAcylation and phosphorylation in cellular regulation.

      Febs Letters
      Acetylation, Acetylglucosamine, metabolism, Acetylglucosaminidase, Animals, Binding Sites, Humans, Mass Spectrometry, Models, Biological, Models, Molecular, N-Acetylglucosaminyltransferases, Phosphorylation, Protein Processing, Post-Translational, physiology, Signal Transduction

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          Abstract

          Ser(Thr)-O-linked beta-N-acetylglucosamine (O-GlcNAc) is a ubiquitous modification of nucleocytoplasmic proteins. Extensive crosstalk exists between O-GlcNAcylation and phosphorylation, which regulates signaling in response to nutrients/stress. The development of novel O-GlcNAc detection and enrichment methods has improved our understanding of O-GlcNAc functions. Mass spectrometry has revealed O-GlcNAc's many interactions with phosphorylation-mediated signaling. However, mechanisms regulating O-GlcNAcylation and phosphorylation are quite different. Phosphorylation is catalyzed by hundreds of distinct kinases. In contrast, in mammals, uridine diphospho-N-acetylglucosamine:polypeptide beta-N-acetylglucosaminyl transferase (OGT) and beta-D-N-acetylglucosaminidase (OGA) are encoded by single highly conserved genes. Both OGT's and OGA's specificities are determined by their transient associations with many other proteins to create a multitude of specific holoenzymes. The extensive crosstalk between O-GlcNAcylation and phosphorylation represents a new paradigm for cellular signaling. Copyright 2010. Published by Elsevier B.V.

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