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Hemorrhagic shock induces endothelial cell apoptosis, which is mediated by factors contained in mesenteric lymph.

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      Abstract

      Trauma-hemorrhagic shock is one of the leading causes of acute respiratory distress syndrome. This syndrome is associated with disruption of the alveolar barrier consisting of both epithelial and endothelial cells, which leads to a major increase in epithelial and microvascular permeability in the lungs. Although alveolar epithelial cell apoptosis has been documented as a contributing factor to this increase in permeability, it is unclear whether endothelial cell apoptosis occurs following trauma-hemorrhagic shock and, if so, the source of factors leading to this process.

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      Affiliations
      [1 ] Department of Surgery, UMDNJ-New Jersey Medical School, Newark, NJ 07103, USA.
      Journal
      Crit. Care Med.
      Critical care medicine
      0090-3493
      0090-3493
      Dec 2004
      : 32
      : 12
      15599152
      00003246-200412000-00015

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