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      Propionibacterium acnes catalase induces increased Th1 immune response in sarcoidosis patients.

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          Abstract

          Propionibacterium acnes is one of the most commonly implicated etiologic agents of sarcoidosis. We screened antigenic proteins from this indigenous bacterium that increase Th1 responses in sarcoidosis patients.

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          Author and article information

          Journal
          Respir Investig
          Respiratory investigation
          Elsevier BV
          2212-5353
          2212-5345
          Jul 2015
          : 53
          : 4
          Affiliations
          [1 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: p.yorozu.pth1@tmd.ac.jp.
          [2 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: askapth1@tmd.ac.jp.
          [3 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: uchida.path@tmd.ac.jp.
          [4 ] Division of Surgical Pathology, Tokyo Medical and Dental University Hospital, Tokyo 113-8510, Japan. Electronic address: akashi.path@tmd.ac.jp.
          [5 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: kakepth1@tmd.ac.jp.
          [6 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: ogawpth1@tmd.ac.jp.
          [7 ] Department of Clinical Engineering, School of Health Sciences, Tokyo University of Technology, Tokyo 144-8650, Japan. Electronic address: jminami@stf.teu.ac.jp.
          [8 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: y.suzuki.pth1@tmd.ac.jp.
          [9 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan; Department of Respiratory Medicine, Japanese Red Cross Medical Center, Tokyo 150-8935, Japan. Electronic address: awanobu0606@hotmail.co.jp.
          [10 ] Department of Integrated Pulmonology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: hfurusawa.pulm@tmd.ac.jp.
          [11 ] Department of Integrated Pulmonology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: miyazaki.pilm@tmd.ac.jp.
          [12 ] Department of Integrated Pulmonology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan. Electronic address: ninase.pulm@tmd.ac.jp.
          [13 ] Department of Human Pathology, Tokyo Medical and Dental University Graduate School, Tokyo 113-8510, Japan; Division of Surgical Pathology, Tokyo Medical and Dental University Hospital, Tokyo 113-8510, Japan. Electronic address: eishi.path@tmd.ac.jp.
          Article
          S2212-5345(15)00033-7
          10.1016/j.resinv.2015.02.005
          26100176
          355ee4d6-139f-4d34-90b2-4b11b0bed4ea
          History

          ELISA,UDP-N-acetylglucosamine pyrophosphorylase,Arginine deiminase,Enzyme-linked immunospot assay

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