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      Gut epithelial barrier dysfunction and innate immune activation predict mortality in treated HIV infection.

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          Abstract

          While inflammation predicts mortality in treated human immunodeficiency virus (HIV) infection, the prognostic significance of gut barrier dysfunction and phenotypic T-cell markers remains unclear.

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          Author and article information

          Journal
          Journal ID (iso-abbrev): J. Infect. Dis.
          Title: The Journal of infectious diseases
          ISSN (Electronic): 1537-6613
          ISSN (Print): 0022-1899
          Publication date (Electronic): Oct 15 2014
          Volume: 210
          Issue: 8
          Affiliations
          [1 ] Department of Medicine, University of California San Francisco.
          [2 ] Department of Medicine, Case Western Reserve University, Cleveland, Ohio.
          [3 ] School of Health and Rehabilitation Sciences, Medical Laboratory Science Division, Ohio State University, Columbus.
          [4 ] Department of Bioengineering and Therapeutic Sciences.
          [5 ] Department of Medicine, University of California San Francisco Department of Epidemiology and Biostatistics, University of California San Francisco.
          [6 ] Department of Epidemiology, The Johns Hopkins University Bloomberg School of Public Health, Baltimore, Maryland.
          [7 ] Department of Epidemiology, The Johns Hopkins University Bloomberg School of Public Health, Baltimore, Maryland Departments of Ophthalmology and Medicine, The Icahn School of Medicine at Mount Sinai, New York, New York.
          Article
          Publisher Item ID: jiu238
          DOI: 10.1093/infdis/jiu238
          PMC ID: 4192038
          PubMed ID: 24755434
          SO-VID: 362df6a5-2bfa-47fd-9f1d-e432e4ac4dbd
          Copyright © © The Author 2014. Published by Oxford University Press on behalf of the Infectious Diseases Society of America. All rights reserved. For Permissions, please e-mail: journals.permissions@oup.com.
          History

          Keywords: CD28,CD38,CD57,D-dimer,HIV,HLA-DR,IL-6,T-cell activation,antiretroviral therapy,cytomegalovirus,gut epithelial cell barrier,hsCRP,immune activation,intestinal fatty acid binding protein (I-FABP),mortality,sCD14,zonulin-1
          Data availability:
          Keywords: CD28, CD38, CD57, D-dimer, HIV, HLA-DR, IL-6, T-cell activation, antiretroviral therapy, cytomegalovirus, gut epithelial cell barrier, hsCRP, immune activation, intestinal fatty acid binding protein (I-FABP), mortality, sCD14, zonulin-1

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