Redox Control of Mitochondrial Calcium Uptake.

Demaurex, Nicolas; Rosselin, Manon

doi:10.1016/j.molcel.2017.02.029

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Redox Control of Mitochondrial Calcium Uptake.

Author(s): Nicolas Demaurex ¹ , Manon Rosselin ²

Publication date (Electronic): Mar 16 2017

Journal: Molecular cell

Publisher: Elsevier BV

Keywords: apoptosis, cell signaling, inflammation, ion channels, metabolism, mitochondria, oxidative stress

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Abstract

Dong et al. (2017) establish how the mitochondrial Ca2+uniporter (MCU) integrates Ca2+and oxidative stress signals by identifying a cysteine residue that controls MCU channel activity, a mechanism causing mitochondrial Ca2+overload and cell death during oxidative stress.

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Journal ID (iso-abbrev): Mol. Cell

Title: Molecular cell

Publisher: Elsevier BV

ISSN (Electronic): 1097-4164

ISSN (Print): 1097-2765

Publication date (Electronic): Mar 16 2017

Volume: 65

Issue: 6

Affiliations

[1 ] Department of Cell Physiology and Metabolism, University of Geneva, 1, rue Michel-Servet, 1211 Geneva 4, Switzerland. Electronic address: nicolas.demaurex@unige.ch.

[2 ] Department of Cell Physiology and Metabolism, University of Geneva, 1, rue Michel-Servet, 1211 Geneva 4, Switzerland.

Article

Publisher Item ID: S1097-2765(17)30166-1

DOI: 10.1016/j.molcel.2017.02.029

PubMed ID: 28306510

SO-VID: 3c77ff11-a10c-476b-a869-1c3e9264a039

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Keywords: cell signaling,oxidative stress,mitochondria,metabolism,ion channels,inflammation,apoptosis

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Keywords: cell signaling, oxidative stress, mitochondria, metabolism, ion channels, inflammation, apoptosis

Redox Control of Mitochondrial Calcium Uptake.

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