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      Prevalence of exposure of heavy metals and their impact on health consequences

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          Most cited references207

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          Is Open Access

          The Nrf2-antioxidant response element signaling pathway and its activation by oxidative stress.

          A major mechanism in the cellular defense against oxidative or electrophilic stress is activation of the Nrf2-antioxidant response element signaling pathway, which controls the expression of genes whose protein products are involved in the detoxication and elimination of reactive oxidants and electrophilic agents through conjugative reactions and by enhancing cellular antioxidant capacity. At the molecular level, however, the regulatory mechanisms involved in mediating Nrf2 activation are not fully understood. It is well established that Nrf2 activity is controlled, in part, by the cytosolic protein Keap1, but the nature of this pathway and the mechanisms by which Keap1 acts to repress Nrf2 activity remain to be fully characterized and are the topics of discussion in this minireview. In addition, a possible role of the Nrf2-antioxidant response element transcriptional pathway in neuroprotection will also be discussed.
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            Electrochemical technologies in wastewater treatment

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              Physico–chemical treatment techniques for wastewater laden with heavy metals

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                Author and article information

                Journal
                Journal of Cellular Biochemistry
                J. Cell. Biochem.
                Wiley
                07302312
                January 2018
                January 2018
                August 02 2017
                : 119
                : 1
                : 157-184
                Affiliations
                [1 ]Institute of Pharmacy, Physiology, and Pharmacology; University of Agriculture; Faisalabad Pakistan
                [2 ]Department of Pharmaceutical Chemistry; Government College University Faisalabad; Faisalabad Pakistan
                Article
                10.1002/jcb.26234
                28643849
                3e58dc92-c0d1-43f3-b7e8-f656dbf0bcbe
                © 2017

                http://doi.wiley.com/10.1002/tdm_license_1.1

                http://onlinelibrary.wiley.com/termsAndConditions#vor

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