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      Global Air Quality and Health Co-benefits of Mitigating Near-Term Climate Change through Methane and Black Carbon Emission Controls

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          Abstract

          Background: Tropospheric ozone and black carbon (BC), a component of fine particulate matter (PM ≤ 2.5 µm in aerodynamic diameter; PM 2.5), are associated with premature mortality and they disrupt global and regional climate.

          Objectives: We examined the air quality and health benefits of 14 specific emission control measures targeting BC and methane, an ozone precursor, that were selected because of their potential to reduce the rate of climate change over the next 20–40 years.

          Methods: We simulated the impacts of mitigation measures on outdoor concentrations of PM 2.5 and ozone using two composition-climate models, and calculated associated changes in premature PM 2.5- and ozone-related deaths using epidemiologically derived concentration–response functions.

          Results: We estimated that, for PM 2.5 and ozone, respectively, fully implementing these measures could reduce global population-weighted average surface concentrations by 23–34% and 7–17% and avoid 0.6–4.4 and 0.04–0.52 million annual premature deaths globally in 2030. More than 80% of the health benefits are estimated to occur in Asia. We estimated that BC mitigation measures would achieve approximately 98% of the deaths that would be avoided if all BC and methane mitigation measures were implemented, due to reduced BC and associated reductions of nonmethane ozone precursor and organic carbon emissions as well as stronger mortality relationships for PM 2.5 relative to ozone. Although subject to large uncertainty, these estimates and conclusions are not strongly dependent on assumptions for the concentration–response function.

          Conclusions: In addition to climate benefits, our findings indicate that the methane and BC emission control measures would have substantial co-benefits for air quality and public health worldwide, potentially reversing trends of increasing air pollution concentrations and mortality in Africa and South, West, and Central Asia. These projected benefits are independent of carbon dioxide mitigation measures. Benefits of BC measures are underestimated because we did not account for benefits from reduced indoor exposures and because outdoor exposure estimates were limited by model spatial resolution.

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          Most cited references54

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          Global and regional climate changes due to black carbon

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            Lung cancer, cardiopulmonary mortality, and long-term exposure to fine particulate air pollution.

            Associations have been found between day-to-day particulate air pollution and increased risk of various adverse health outcomes, including cardiopulmonary mortality. However, studies of health effects of long-term particulate air pollution have been less conclusive. To assess the relationship between long-term exposure to fine particulate air pollution and all-cause, lung cancer, and cardiopulmonary mortality. Vital status and cause of death data were collected by the American Cancer Society as part of the Cancer Prevention II study, an ongoing prospective mortality study, which enrolled approximately 1.2 million adults in 1982. Participants completed a questionnaire detailing individual risk factor data (age, sex, race, weight, height, smoking history, education, marital status, diet, alcohol consumption, and occupational exposures). The risk factor data for approximately 500 000 adults were linked with air pollution data for metropolitan areas throughout the United States and combined with vital status and cause of death data through December 31, 1998. All-cause, lung cancer, and cardiopulmonary mortality. Fine particulate and sulfur oxide--related pollution were associated with all-cause, lung cancer, and cardiopulmonary mortality. Each 10-microg/m(3) elevation in fine particulate air pollution was associated with approximately a 4%, 6%, and 8% increased risk of all-cause, cardiopulmonary, and lung cancer mortality, respectively. Measures of coarse particle fraction and total suspended particles were not consistently associated with mortality. Long-term exposure to combustion-related fine particulate air pollution is an important environmental risk factor for cardiopulmonary and lung cancer mortality.
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              Long-term ozone exposure and mortality.

              Although many studies have linked elevations in tropospheric ozone to adverse health outcomes, the effect of long-term exposure to ozone on air pollution-related mortality remains uncertain. We examined the potential contribution of exposure to ozone to the risk of death from cardiopulmonary causes and specifically to death from respiratory causes. Data from the study cohort of the American Cancer Society Cancer Prevention Study II were correlated with air-pollution data from 96 metropolitan statistical areas in the United States. Data were analyzed from 448,850 subjects, with 118,777 deaths in an 18-year follow-up period. Data on daily maximum ozone concentrations were obtained from April 1 to September 30 for the years 1977 through 2000. Data on concentrations of fine particulate matter (particles that are < or = 2.5 microm in aerodynamic diameter [PM(2.5)]) were obtained for the years 1999 and 2000. Associations between ozone concentrations and the risk of death were evaluated with the use of standard and multilevel Cox regression models. In single-pollutant models, increased concentrations of either PM(2.5) or ozone were significantly associated with an increased risk of death from cardiopulmonary causes. In two-pollutant models, PM(2.5) was associated with the risk of death from cardiovascular causes, whereas ozone was associated with the risk of death from respiratory causes. The estimated relative risk of death from respiratory causes that was associated with an increment in ozone concentration of 10 ppb was 1.040 (95% confidence interval, 1.010 to 1.067). The association of ozone with the risk of death from respiratory causes was insensitive to adjustment for confounders and to the type of statistical model used. In this large study, we were not able to detect an effect of ozone on the risk of death from cardiovascular causes when the concentration of PM(2.5) was taken into account. We did, however, demonstrate a significant increase in the risk of death from respiratory causes in association with an increase in ozone concentration. 2009 Massachusetts Medical Society
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                Author and article information

                Journal
                Environ Health Perspect
                Environ. Health Perspect
                EHP
                Environmental Health Perspectives
                National Institute of Environmental Health Sciences
                0091-6765
                1552-9924
                14 March 2012
                June 2012
                : 120
                : 6
                : 831-839
                Affiliations
                [1 ]U.S. Environmental Protection Agency, Washington, DC, USA
                [2 ]Department of Environmental Health, Harvard School of Public Health, Boston, Massachusetts, USA
                [3 ]NASA Goddard Institute for Space Studies and Columbia Earth Institute, Columbia University, New York, New York, USA
                [4 ]International Institute for Applied Systems Analysis, Laxenburg, Austria
                [5 ]European Commission, Joint Research Centre, Ispra, Italy
                [6 ]Stockholm Environment Institute, Environment Department, University of York, York, United Kingdom
                [7 ]Department of Economics, Middlebury College, Middlebury, Vermont, USA
                [8 ]Environmental Sciences and Engineering Department, Gillings School of Global Public Health, University of North Carolina–Chapel Hill, Chapel Hill, North Carolina, USA
                [9 ]Environmental Research Group, King’s College London, London, United Kingdom
                [10 ]United Nations Environment Programme, Nairobi, Kenya
                [11 ]Scripps Institution of Oceanography, University of California–San Diego, San Diego, California, USA
                Author notes
                Address correspondence to S.C. Anenberg, U.S. Environmental Protection Agency, 1200 Pennsylvania Ave. NW, MC-6301A, Washington, DC 20460 USA. Telephone: (202) 564-2065. Fax: (202) 564-1543. E-mail: anenberg.susan@ 123456epa.gov
                [*]

                Current address: Eurasia Institute of Earth Sciences, Istanbul Technical University, Istanbul, Turkey.

                Article
                ehp.1104301
                10.1289/ehp.1104301
                3385429
                22418651
                5204f805-2592-4da5-b533-007ce89233f6
                Copyright @ 2012

                This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

                History
                : 04 August 2011
                : 14 March 2012
                Categories
                Research

                Public health
                outdoor air,particulate matter,air quality,health impact analysis,climate change
                Public health
                outdoor air, particulate matter, air quality, health impact analysis, climate change

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