Adipose Tissue Drives Response to Ischemia-Reperfusion Injury in a Murine Pressure Sore Model.

There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

Abstract

Ischemia-reperfusion injury contributes significantly to the pathogenesis of chronic wounds such as pressure sores and diabetic foot ulcers. The authors' laboratory has previously developed a cyclical murine ischemia-reperfusion injury model. The authors here use this model to determine factors underlying tissue response to ischemia-reperfusion injury.

Related collections

Author and article information

Journal

Journal ID (iso-abbrev): Plast. Reconstr. Surg.

Title: Plastic and reconstructive surgery

Publisher: Ovid Technologies (Wolters Kluwer Health)

ISSN (Electronic): 1529-4242

ISSN (Print): 0032-1052

Publication date (Electronic): May 2017

Volume: 139

Issue: 5

Affiliations

[1 ] Chicago, Ill. From the Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Division of Plastic and Reconstructive Surgery, Northwestern University, Feinberg School of Medicine.

Article

Publisher Item ID: 00006534-201705000-00026

DOI: 10.1097/PRS.0000000000003271

PubMed ID: 28445367

SO-VID: 576dcfdc-af09-4c93-9507-8f74631fc276

History

Data availability:

Comments

Comment on this article

scite_

Cited by 3

See all cited by