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      Landau Theory of Adaptive Integration in Computational Intelligence

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          Abstract

          Computational Intelligence (CI) is a sub-branch of Artificial Intelligence paradigm focusing on the study of adaptive mechanisms to enable or facilitate intelligent behavior in complex and changing environments. There are several paradigms of CI [like artificial neural networks, evolutionary computations, swarm intelligence, artificial immune systems, fuzzy systems and many others], each of these has its origins in biological systems [biological neural systems, natural Darwinian evolution, social behavior, immune system, interactions of organisms with their environment]. Most of those paradigms evolved into separate machine learning (ML) techniques, where probabilistic methods are used complementary with CI techniques in order to effectively combine elements of learning, adaptation, evolution and Fuzzy logic to create heuristic algorithms that are, in some sense, intelligent. The current trend is to develop consensus techniques, since no single machine learning algorithms is superior to others in all possible situations. In order to overcome this problem several meta-approaches were proposed in ML focusing on the integration of results from different methods into single prediction. We discuss here the Landau theory for the nonlinear equation that can describe the adaptive integration of information acquired from an ensemble of independent learning agents. The influence of each individual agent on other learners is described similarly to the social impact theory. The final decision outcome for the consensus system is calculated using majority rule in the stationary limit, yet the minority solutions can survive inside the majority population as the complex intermittent clusters of opposite opinion.

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          Most cited references27

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          Reinforcement learning of motor skills with policy gradients.

          Autonomous learning is one of the hallmarks of human and animal behavior, and understanding the principles of learning will be crucial in order to achieve true autonomy in advanced machines like humanoid robots. In this paper, we examine learning of complex motor skills with human-like limbs. While supervised learning can offer useful tools for bootstrapping behavior, e.g., by learning from demonstration, it is only reinforcement learning that offers a general approach to the final trial-and-error improvement that is needed by each individual acquiring a skill. Neither neurobiological nor machine learning studies have, so far, offered compelling results on how reinforcement learning can be scaled to the high-dimensional continuous state and action spaces of humans or humanoids. Here, we combine two recent research developments on learning motor control in order to achieve this scaling. First, we interpret the idea of modular motor control by means of motor primitives as a suitable way to generate parameterized control policies for reinforcement learning. Second, we combine motor primitives with the theory of stochastic policy gradient learning, which currently seems to be the only feasible framework for reinforcement learning for humanoids. We evaluate different policy gradient methods with a focus on their applicability to parameterized motor primitives. We compare these algorithms in the context of motor primitive learning, and show that our most modern algorithm, the Episodic Natural Actor-Critic outperforms previous algorithms by at least an order of magnitude. We demonstrate the efficiency of this reinforcement learning method in the application of learning to hit a baseball with an anthropomorphic robot arm.
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            Mathematical modeling of planar cell polarity to understand domineering nonautonomy.

            Planar cell polarity (PCP) signaling generates subcellular asymmetry along an axis orthogonal to the epithelial apical-basal axis. Through a poorly understood mechanism, cell clones that have mutations in some PCP signaling components, including some, but not all, alleles of the receptor frizzled, cause polarity disruptions of neighboring wild-type cells, a phenomenon referred to as domineering nonautonomy. Here, a contact-dependent signaling hypothesis, derived from experimental results, is shown by reaction-diffusion, partial differential equation modeling and simulation to fully reproduce PCP phenotypes, including domineering nonautonomy, in the Drosophila wing. The sufficiency of this model and the experimental validation of model predictions reveal how specific protein-protein interactions produce autonomy or domineering nonautonomy.
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              Interaction between Wingless and Notch signaling pathways mediated by dishevelled.

              In Drosophila, the Wingless and Notch signaling pathways function in m any of the same developmental patterning events. Genetic analysis demonstrates that the dishevelled gene, which encodes a molecule previously implicated in implementation of the Winglass signal, interacts antagonistically with Notch and one of its known ligands, Delta. A direct physical interaction between Dishevelled and the Notch carboxyl terminus, distal to the cdc10/ankyrin repeats, suggests a mechanism for this interaction. It is proposed that Dishevelled, in addition to transducing the Wingless signal, blocks Notch signaling directly, thus providing a molecular mechanism for the inhibitory cross talk observed between these pathways.
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                Author and article information

                Journal
                09 June 2010
                Article
                1006.1828
                661bd871-ea06-490a-a9cd-ad4c81be22d9

                http://arxiv.org/licenses/nonexclusive-distrib/1.0/

                History
                Custom metadata
                19 pages
                stat.ML cs.AI nlin.AO q-bio.NC q-bio.PE

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