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      Advanced glycation end products accelerate arteriosclerosis after renal transplantation through the AGE/RAGE/ILK pathway.

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          Abstract

          The effects of advanced glycation end products (AGEs) on arteriosclerosis (AS) after kidney transplantation and the molecular mechanisms involved remain unclear.

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          Author and article information

          Journal
          Exp. Mol. Pathol.
          Experimental and molecular pathology
          Elsevier BV
          1096-0945
          0014-4800
          Oct 2015
          : 99
          : 2
          Affiliations
          [1 ] Department of Urology, The First Affiliated Hospital of Nanjing Medical University, 300 Guangzhou Road, Nanjing, Jiangsu Province 210029, China.
          [2 ] Department of Urology, The First Affiliated Hospital of Nanjing Medical University, 300 Guangzhou Road, Nanjing, Jiangsu Province 210029, China; Department of Urology, Huai'an First People's Hospital, 6 Beijing West Road, Huai'an, Jiangsu Province 223300, China.
          [3 ] Department of Urology, Suzhou Municipal Hospital, 26 Daoqian Road, Suzhou, Jiangsu Province 215001, China.
          [4 ] Department of Urology, The First Affiliated Hospital of Nanjing Medical University, 300 Guangzhou Road, Nanjing, Jiangsu Province 210029, China. Electronic address: Lancetgu@aliyun.com.cn.
          [5 ] Department of Urology, The First Affiliated Hospital of Nanjing Medical University, 300 Guangzhou Road, Nanjing, Jiangsu Province 210029, China. Electronic address: tanruoyun112@vip.sina.com.
          Article
          S0014-4800(15)00157-4
          10.1016/j.yexmp.2015.07.009
          26210487
          6c6a307f-3e74-43b2-964a-4c6c4f028885
          History

          AGEs,Arteriosclerosis,ILK,Kidney transplantation,Osteoblast,RAGE,Trans-differentiation,VSMCs

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