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      CLC-3 deficiency leads to phenotypes similar to human neuronal ceroid lipofuscinosis.

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          Abstract

          CLC-3 is a member of the CLC chloride channel family and is widely expressed in mammalian tissues. To determine the physiological role of CLC-3, we generated CLC-3-deficient mice (Clcn3-/- ) by targeted gene disruption.

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          Author and article information

          Journal
          Genes Cells
          Genes to cells : devoted to molecular & cellular mechanisms
          1356-9597
          1356-9597
          Jun 2002
          : 7
          : 6
          Affiliations
          [1 ] Homeostasis Medicine and Nephrology, Graduate School, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyo Tokyo 113-8519, Japan.
          Article
          539
          10.1046/j.1365-2443.2002.00539
          12059962
          6cc0a181-b50c-4bae-9462-3f7b488724fb
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