The endoplasmic reticulum stress-C/EBP homologous protein pathway-mediated apoptosis in macrophages contributes to the instability of atherosclerotic plaques.
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Abstract
To elucidate whether and how the endoplasmic reticulum (ER) stress-C/EBP homologous
protein (CHOP) pathway in macrophages is involved in the rupture of atherosclerotic
plaques.