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      Prevalence of Hazardous Occupational Noise Exposure, Hearing Loss, and Hearing Protection Usage Among a Representative Sample of Working Canadians

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          Abstract

          Objective:

          The aim of this study was to estimate the prevalence of hearing loss (HL), self-reported occupational noise exposure, and hearing protection usage among Canadians.

          Methods:

          In-person household interviews were conducted with 3666 participants, aged 16 to 79 years (1811 males) with 94% completing audiometry and distortion-product otoacoustic emission (DPOAE) evaluations. Occupational noise exposure was defined as hazardous when communicating with coworkers at an arm's length distance required speaking in a raised voice.

          Results:

          An estimated 42% of respondents reported hazardous occupational noise exposure; 10 years or more was associated with HL regardless of age, sex or education. Absent DPOAEs, tinnitus, and the Wilson audiometric notch were significantly more prevalent in hazardous workplace noise-exposed workers than in nonexposed. When mandatory, 80% reported wearing hearing protection.

          Conclusions:

          These findings are consistent with other industrialized countries, underscoring the need for ongoing awareness of noise-induced occupational HL.

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          Most cited references79

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          Tinnitus with a normal audiogram: physiological evidence for hidden hearing loss and computational model.

          Ever since Pliny the Elder coined the term tinnitus, the perception of sound in the absence of an external sound source has remained enigmatic. Traditional theories assume that tinnitus is triggered by cochlear damage, but many tinnitus patients present with a normal audiogram, i.e., with no direct signs of cochlear damage. Here, we report that in human subjects with tinnitus and a normal audiogram, auditory brainstem responses show a significantly reduced amplitude of the wave I potential (generated by primary auditory nerve fibers) but normal amplitudes of the more centrally generated wave V. This provides direct physiological evidence of "hidden hearing loss" that manifests as reduced neural output from the cochlea, and consequent renormalization of neuronal response magnitude within the brainstem. Employing an established computational model, we demonstrate how tinnitus could arise from a homeostatic response of neurons in the central auditory system to reduced auditory nerve input in the absence of elevated hearing thresholds.
            • Record: found
            • Abstract: found
            • Article: not found

            Classification and epidemiology of tinnitus.

            One third of all adults report experiencing tinnitus at some time in their lives. Ten percent to 15% have prolonged tinnitus requiring medical evaluation. Classification of tinnitus requires a thorough history and physical examination, supplemented by appropriate diagnostic tests. Tinnitus can be categorized according to its qualities (as described by the patient and matched on the audiometer) and its clinical type (as suggested by probable etiology). Audiologic testing, tinnitus analysis, and occasional radiologic studies assist with classification and direction of treatment planning. The THI is another method of classification that can facilitate the precise monitoring of a patient's progress. By using these tools and standardizing the language, tinnitus studies around the world can become more comparable and patients can be better monitored for treatment response.
              • Record: found
              • Abstract: not found
              • Article: not found

              Preferred Method For Clinical Determination Of Pure-Tone Thresholds

                Author and article information

                Journal
                J Occup Environ Med
                J. Occup. Environ. Med
                JOEM
                Journal of Occupational and Environmental Medicine
                Lippincott Williams & Wilkins
                1076-2752
                1536-5948
                January 2017
                20 December 2016
                : 59
                : 1
                : 92-113
                Affiliations
                Health Effects and Assessment Division, Health Canada (Drs Feder, Michaud, McNamee), Audiology & Speech-language Pathology Program, Faculty of Health Sciences, University of Ottawa, Ottawa, Ontario (Dr Fitzpatrick), Occupational & Environmental Health, School of Population & Public Health, University of British Columbia, Vancouver, British Columbia (Dr Davies), and École d’orthophonie et d’audiologie, Université de Montréal, Faculté de médecine, Montréal, Québec, Canada (Dr Leroux).
                Author notes
                Address correspondence to: Katya Feder, PhD, Health Effects and Assessment Division, Health Canada, 775 Brookfield Road, Ottawa, ON K1A 1C1, Canada ( Katya.Feder@ 123456hc-sc.gc.ca ).
                Article
                JOEM-16-5972 00015
                10.1097/JOM.0000000000000920
                5704673
                28045804
                7fd6b96c-27ee-4cd9-83a0-0156611af512
                Copyright © 2016 American College of Occupational and Environmental Medicine

                This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0

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