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      Modeling Transitions between Responsive and Resistant States in Breast Cancer with Application to Therapy Optimization

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      bioRxiv

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          Abstract

          We present a mathematical model that captures the transitions among three experimentally observed estrogen sensitivity phenotypes in breast cancer cells. Based on this model, a population-level model is created and used to explore the optimization of a therapeutic protocol

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          Author and article information

          Journal
          bioRxiv
          November 10 2015
          Article
          10.1101/031245
          86073c1b-30fc-4679-a7e0-faed377e6dec
          © 2015
          History

          Cell biology,Molecular biology
          Cell biology, Molecular biology

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