15
views
0
recommends
+1 Recommend
1 collections
    0
    shares
      • Record: found
      • Abstract: found
      • Article: found
      Is Open Access

      Paraplejia por hipopotasemia secundaria a diarrea aguda Translated title: Hipokalemic paraplegia due to acute diarrhea

      brief-report

      Read this article at

      SciELO
          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Abstract

          La hipopotasemia puede dar una clínica variada pero es frecuente que curse de forma asintomática o con síntomas inespecíficos si es de escasa cantidad. En nuestro medio las causas de la hipopotasemia son variadas pero una de las etiologías más frecuentes en paises en vías de desarrollo son los cuadros diarreicos. Presentamos un cuadro de hipopotasemia severa debida a diarrea que se manifestó con intensa clínica neurológica pero que mejoró espectacularmente con tratamiento habitual.

          Translated abstract

          Hipokalemia can give a variety of sintomatology but more often courses without it or with inespecifyc clinical manifestations. In our enviroment the etiology of hipokalemia is wide but one of the most common causes in third world countries are diarrheas. We describe a case of severe hipokalemia due to acute diarrhea which was maniffested with severe neurologyc sintoms but improves with conventional treatment.

          Related collections

          Most cited references13

          • Record: found
          • Abstract: found
          • Article: not found

          Pathophysiology of potassium absorption and secretion by the human intestine.

          When normal people ingest 90 mEq/day of K+ in their diet, they absorb about 90% of intake (81 mEq) and excrete an equivalent amount of K+ in the urine. Normal fecal K+ excretion averages about 9 mEq/day. The vast majority of intestinal K+ absorption occurs in the small intestine; the contribution of the normal colon to net K+ absorption and secretion is trivial. K+ is absorbed or secreted mainly by passive mechanisms; the rectum and perhaps the sigmoid colon have the capacity to actively secrete K+, but the quantitative and physiological significance of this active secretion is uncertain. Hyperaldosteronism increases fecal K+ excretion by about 3 mEq/day in people with otherwise normal intestinal tracts. Cation exchange resin by mouth can increase fecal K+ excretion to 40 mEq/day. The absorptive mechanisms of K+ are not disturbed by diarrhea per se, but fecal K+ losses are increased in diarrheal diseases by unabsorbed anions (which obligate K+), by electrochemical gradients secondary to active chloride secretion, and probably by secondary hyperaldosteronism. In diarrhea, total body K+ can be reduced by two mechanisms: loss of muscle mass because of malnutrition and reduced net absorption of K+; only the latter causes hypokalemia. Balance studies in patients with diarrhea are exceedingly rare, but available data emphasize an important role for dietary K+ intake, renal K+ excretion, and fecal K+ losses in determining whether or not a patient develops hypokalemia. The paradoxical negative K+ balance induced by ureterosigmoid anastomosis is described. The concept that fecal K+ excretion is markedly elevated in patients with uremia as an intestinal adaptation to prevent hyperkalemia is analyzed; we conclude that the data do not convincingly show the existence of a major intestinal adaptive response to chronic renal failure.
            • Record: found
            • Abstract: not found
            • Article: not found

            Flaccid Quadriparesis Associated With Yersinia Enterocolitis-Induced Hypokalemia

              • Record: found
              • Abstract: not found
              • Article: not found

              hipocaliémica tirotóxica: 18 casos por diferentes formas de tirotoxicosis

              (1995)

                Author and article information

                Contributors
                Role: ND
                Role: ND
                Role: ND
                Role: ND
                Role: ND
                Journal
                ami
                Anales de Medicina Interna
                An. Med. Interna (Madrid)
                Arán Ediciones, S. L. (Madrid )
                0212-7199
                February 2002
                : 19
                : 2
                : 34-36
                Affiliations
                [1 ] Hospital Clínico San Carlos de Madrid Spain
                [2 ] Hospital Clínico San Carlos de Madrid Spain
                Article
                S0212-71992002000200006
                10.4321/s0212-71992002000200006
                8a583cb6-9d18-4138-b2cf-b43be72b7d8f

                http://creativecommons.org/licenses/by/4.0/

                History
                Categories
                MEDICINE, GENERAL & INTERNAL

                Internal medicine
                Hipokalemia,Paraplegia,Diarrhea,Hipopotasemia,Paraplejia,Diarrea
                Internal medicine
                Hipokalemia, Paraplegia, Diarrhea, Hipopotasemia, Paraplejia, Diarrea

                Comments

                Comment on this article

                Related Documents Log