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      Updated Understanding of the Degenerative Disc Diseases - Causes Versus Effects - Treatments, Studies and Hypothesis

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          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Abstract

          Background

          In this review we survey medical treatments and research strategies, and we discuss why they have failed to cure degenerative disc diseases or even slow down the degenerative process.

          Objective

          We seek to stimulate discussion with respect to changing the medical paradigm associated with treatments and research applied to degenerative disc diseases.

          Method Proposal

          We summarize a Biological Transformation therapy for curing chronic inflammations and degenerative disc diseases, as was previously described in the book Biological Transformations controlled by the Mind Volume 1.

          Preliminary Studies

          A single-patient case study is presented that documents complete recovery from an advanced lumbar bilateral discopathy and long-term hypertrophic chronic rhinitis by application of the method proposed.

          Conclusion

          Biological transformations controlled by the mind can be applied by men and women in order to improve their quality of life and cure degenerative disc diseases and chronic inflammations illnesses.

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          The benefits of being present: Mindfulness and its role in psychological well-being.

          Kirk Brown, Richard M. Ryan (2003)
          Mindfulness is an attribute of consciousness long believed to promote well-being. This research provides a theoretical and empirical examination of the role of mindfulness in psychological well-being. The development and psychometric properties of the dispositional Mindful Attention Awareness Scale (MAAS) are described. Correlational, quasi-experimental, and laboratory studies then show that the MAAS measures a unique quality of consciousness that is related to a variety of well-being constructs, that differentiates mindfulness practitioners from others, and that is associated with enhanced self-awareness. An experience-sampling study shows that both dispositional and state mindfulness predict self-regulated behavior and positive emotional states. Finally, a clinical intervention study with cancer patients demonstrates that increases in mindfulness over time relate to declines in mood disturbance and stress.
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            Effects of mindfulness on psychological health: a review of empirical studies.

            Shian-Ling Keng, Moria Smoski, Clive Robins (2011)
            Within the past few decades, there has been a surge of interest in the investigation of mindfulness as a psychological construct and as a form of clinical intervention. This article reviews the empirical literature on the effects of mindfulness on psychological health. We begin with a discussion of the construct of mindfulness, differences between Buddhist and Western psychological conceptualizations of mindfulness, and how mindfulness has been integrated into Western medicine and psychology, before reviewing three areas of empirical research: cross-sectional, correlational research on the associations between mindfulness and various indicators of psychological health; intervention research on the effects of mindfulness-oriented interventions on psychological health; and laboratory-based, experimental research on the immediate effects of mindfulness inductions on emotional and behavioral functioning. We conclude that mindfulness brings about various positive psychological effects, including increased subjective well-being, reduced psychological symptoms and emotional reactivity, and improved behavioral regulation. The review ends with a discussion on mechanisms of change of mindfulness interventions and suggested directions for future research. Copyright © 2011 Elsevier Ltd. All rights reserved.
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              Circulating Mitochondrial DAMPs Cause Inflammatory Responses to Injury

              Rui-Qin Zhang, Mustafa Raoof, Yu Chen … (2010)
              Injury causes a systemic inflammatory response syndrome (SIRS) clinically much like sepsis 1. Microbial pathogen-associated molecular patterns (PAMPs) activate innate immunocytes through pattern recognition receptors 2. Similarly, cellular injury can release endogenous damage-associated molecular patterns (DAMPs) that activate innate immunity 3. Mitochondria are evolutionary endosymbionts that were derived from bacteria 4 and so might bear bacterial molecular motifs. We show here that injury releases mitochondrial DAMPs (MTD) into the circulation with functionally important immune consequences. MTD include formyl peptides and mitochondrial DNA. These activate human neutrophils (PMN) through formyl peptide receptor-1 and TLR9 respectively. MTD promote PMN Ca2+ flux and phosphorylation of MAP kinases, thus leading to PMN migration and degranulation in vitro and in vivo. Circulating MTD can elicit neutrophil-mediated organ injury. Cellular disruption by trauma releases mitochondrial DAMPs with evolutionarily conserved similarities to bacterial PAMPs into the circulation. These can then signal through identical innate immune pathways to create a sepsis-like state. The release of such mitochondrial ‘enemies within’ by cellular injury is a key link between trauma, inflammation and SIRS.
              Article 0 comments Cited 580 times – based on 0 reviews     Review now
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                Author and article information

                Journal
                Journal ID (nlm-ta): Curr Genomics
                Journal ID (iso-abbrev): Curr Genomics
                Journal ID (publisher-id): CG
                Title: Current Genomics
                Publisher: Bentham Science Publishers
                ISSN (Print): 1389-2029
                ISSN (Electronic): 1875-5488
                Publication date (Electronic): September 2020
                Publication date (Print): September 2020
                Volume: 21
                Issue: 6
                Pages: 464-477
                Affiliations
                Romanian Television, TVR Cluj, 160 Donath Street, Cluj-Napoca, CJ 400293, , Romania ; Department of Biological Sciences, Salem University , Salem, , WV , USA; Institute of Physiologically Active Compounds Russian Academy of Sciences, Chernogolovka, , 142432 , Russia; Department of Spinal Pathology and Neurosurgery, Turner Scientific and Research Institute for Children’s Orthopedics, Street Parkovskya 64-68, Pushkin, Saint-Petersburg, 196603, , Russia ; Center for Cardiovascular Research (CCR), Institute of Pharmacology, Charite, Universitaetsmedizin Berlin, AG: Theuring, Hessische Strasse 2-4, D-10115, Berlin , Germany, Obie-Medical Corporate International LTD., Academic Section, 28 Oranye Street (American Quarters), Onitsha, Anambra State, Nigeria; I. M. Sechenov First Moscow State Medical University (Sechenov University), Trubetskaya Str., 8, Bld. 2, Moscow, , 119991 , Russia; Stress Relief and Memory Training Center, 3101 Ocean Pkwy, Suite 1A, Brooklyn, , NY, , 11235 , USA; National Research University Higher School of Economics, 20 Myasnitskaya Street, Moscow, , 101000 , Russia; Research Institute of Human Morphology, 3, Tsyurupy Str., Moscow, , 117418 , Russia; GALLY International Research Institute, 7733 Louis Pasteur Drive, #330, San Antonio, , TX, 78229 , USA
                Author notes
                [* ]Address correspondence to this author at the GALLY International Research Institute, 7733 Louis Pasteur Drive, #330, San Antonio, TX, 78229 USA; Tel: +440-263-7461; E-mails: aliev03@ 123456gmail.com and cobalt55@ 123456gallyinternational.com
                Article
                Publisher ID: CG-21-464
                DOI: 10.2174/1389202921999200407082315
                PMC ID: 7536794
                PubMed ID: 33093808
                SO-VID: 8ae671d8-cd1c-4e92-8394-f4f19d2a7466
                Copyright © © 2020 Bentham Science Publishers
                License:

                This is an open access article licensed under the terms of the Creative Commons Attribution-Non-Commercial 4.0 International Public License (CC BY-NC 4.0) ( https://creativecommons.org/licenses/by-nc/4.0/legalcode), which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

                History
                Date received : 25 July 2019
                Date revision received : 20 August 2019
                Date accepted : 16 March 2020
                Categories
                Subject: Article

                ScienceOpen disciplines: Genetics
                Keywords: biology,biological transformations,genome,mitochondria,degenerative disc disease,neurology,seminal secretions
                Data availability:
                ScienceOpen disciplines: Genetics
                Keywords: biology, biological transformations, genome, mitochondria, degenerative disc disease, neurology, seminal secretions

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