18
views
0
recommends
+1 Recommend
0 collections
    0
    shares
      • Record: found
      • Abstract: not found
      • Article: not found

      Non-thermal plasma prevents progression of endometriosis in mice

      Read this article at

      ScienceOpenPublisher
      Bookmark
          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Related collections

          Most cited references33

          • Record: found
          • Abstract: found
          • Article: not found

          Pathogenesis and pathophysiology of endometriosis.

          Originally described over three hundred years ago, endometriosis is classically defined by the presence of endometrial glands and stroma in extrauterine locations. Endometriosis is an inflammatory, estrogen-dependent condition associated with pelvic pain and infertility. This work reviews the disease process from theories regarding origin to the molecular basis for disease sequelae. A thorough understanding of the histopathogenesis and pathophysiology of endometriosis is essential to the development of novel diagnostic and treatment approaches for this debilitating condition. Copyright © 2012 American Society for Reproductive Medicine. All rights reserved.
            Bookmark
            • Record: found
            • Abstract: found
            • Article: not found

            Endometriosis.

            Endometriosis is an oestrogen-dependent disorder that can result in substantial morbidity, including pelvic pain, multiple operations, and infertility. New findings on the genetics, the possible roles of the environment and the immune system, and intrinsic abnormalities in the endometrium of affected women and secreted products of endometriotic lesions have given insight into the pathogenesis of this disorder and serve as the background for new treatments for disease-associated pain and infertility. Affected women are at higher risk than the general female population of developing ovarian cancer, and they also may be at increased risk of breast and other cancers as well as autoimmune and atopic disorders. Clinicians should assess and follow up affected women for these and other associated disorders. There will probably be a new repertoire of approaches for treatment and perhaps cure of this enigmatic disorder in the near future.
              Bookmark
              • Record: found
              • Abstract: found
              • Article: not found

              Persistent oxidative stress in cancer.

              DNA of cancers such as renal cell carcinoma and mammary invasive ductal carcinoma, is persistently exposed to more oxidative stress than that of adjacent normal tissue. We suggest that the concept of 'persistent oxidative stress in cancer' may open up a new research area, explaining part of the characteristic tumor biology of cancer such as activated transcription factors and proto-oncogenes, genomic instability, chemotherapy-resistance, invasion and metastasis.
                Bookmark

                Author and article information

                Journal
                Free Radical Research
                Free Radical Research
                Informa UK Limited
                1071-5762
                1029-2470
                October 02 2016
                August 08 2016
                October 02 2016
                : 50
                : 10
                : 1131-1139
                Affiliations
                [1 ] Department of Pathology and Biological Responses, Nagoya University Graduate School of Medicine, Nagoya, Japan;
                [2 ] Department of Obstetrics and Gynecology, Nagoya University Graduate School of Medicine, Nagoya, Japan;
                [3 ] Center for Advanced Medicine and Clinical Research, Nagoya University Hospital, Nagoya, Japan;
                [4 ] Plasma Nanotechnology Research Center, Nagoya University, Nagoya, Japan;
                [5 ] Sydney Medical School, The University of Sydney, Sydney, Australia
                Article
                10.1080/10715762.2016.1211273
                98ac4795-a69c-4ad9-bd76-fc535df563d2
                © 2016
                History

                Comments

                Comment on this article