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      Catecholamines in Blood and Myocardial Tissue in Experimental Subarachnoidal Hemorrhage

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          Abstract

          The induction of experimental subarachnoidal hemorrhage (ESH) in the dog results in a rise in the adrenaline plasma concentration. The changes in plasma noradrenaline are biphasic: an ‘early noradrenaline gap’ during the first 2 min is followed by a ‘late noradrenaline peak’. It is likely that adrenaline is chiefly concerned with the systemic reactions occurring after ESH, although it can have additional effects at the myocardial level. There is a nonlinear relation between catecholamine concentration changes in tissue and plasma and the myocardial reaction to ESH. This can be explained by the complex interaction of the different factors regulating the level of the catecholamines in blood and tissues.

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          Author and article information

          Journal
          CRD
          Cardiology
          10.1159/issn.0008-6312
          Cardiology
          S. Karger AG
          0008-6312
          1421-9751
          1973
          1973
          29 October 2008
          : 58
          : 4
          : 229-237
          Affiliations
          Laboratory of Human Biochemistry, RUCA, University of Antwerp and Unit for Cardiovascular Research, Born-Bunge Foundation, Antwerp
          Article
          169638 Cardiology 1973;58:229–237
          10.1159/000169638
          4784688
          9a108dd9-473e-4230-b94f-3b7a60f9b70c
          © 1973 S. Karger AG, Basel

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          History
          Page count
          Pages: 9
          Categories
          Paper

          General medicine,Neurology,Cardiovascular Medicine,Internal medicine,Nephrology
          Dog,Adrenaline in blood and tissue,Catecholamines,Experimental sub-arachnoidal hemorrhage,Noradrenaline

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