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      Chronic refractory venous ulcer exacerbated by a congenital pelvic arteriovenous malformation successfully treated by transarterial Onyx embolization.

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          Abstract

          Congenital arteriovenous malformations (AVMs) are an important but often neglected cause of lower extremity venous hypertension. A case of a chronic refractory venous stasis ulcer of the lower extremity exacerbated by a pelvic AVM is presented. Healing of the ulcer was achieved at 2 months without recurrence at 1 year after superselective ethylene vinyl alcohol copolymer (Onyx 34; ev3, Covidien, Plymouth, Minn) embolization of the AVM nidus. Chronic venous arterialization should be considered in cases of refractory, nonhealing venous ulcers. Embolotherapy in addition to standard of care therapy can be a therapeutic measure. Modification of the Clinical, Etiology, Anatomy, and Pathophysiology (CEAP) classification to include AVMs as a contributing pathophysiologic process is suggested.

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          Author and article information

          Journal
          J Vasc Surg Venous Lymphat Disord
          Journal of vascular surgery. Venous and lymphatic disorders
          Elsevier BV
          2213-3348
          May 2017
          : 5
          : 3
          Affiliations
          [1 ] Vascular Anomalies & Malformations Program (VAMP) at the Division of Vascular Surgery, Department of Surgery, Rutgers Robert Wood Johnson Medical School, Robert Wood Johnson University Hospital, Bristol-Myers Squibb Children's Hospital, New Brunswick, NJ. Electronic address: nn207@rutgers.edu.
          [2 ] Rutgers Robert Wood Johnson Medical School, New Brunswick, NJ.
          [3 ] Director for the Center for Wound Healing at Robert Wood Johnson University Hospital, New Brunswick, NJ.
          [4 ] Division of Vascular Surgery, Department of Surgery, The Center for Wound Healing, Rutgers Robert Wood Johnson Medical School, Robert Wood Johnson University Hospital, New Brunswick, NJ.
          Article
          S2213-333X(17)30004-5
          10.1016/j.jvsv.2016.12.001
          28411709
          9bedf56d-9bff-4d34-8b52-ad41cbe0f526
          History

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