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An interferon-gamma-producing Th1 subset is the major source of IL-17 in experimental autoimmune encephalitis.
2007-01-31
Animals, Cell Proliferation, drug effects, Central Nervous System, pathology, Cytokines, metabolism, Disease Models, Animal, Encephalomyelitis, Autoimmune, Experimental, chemically induced, Flow Cytometry, methods, Glycoproteins, Interferon-gamma, pharmacology, Interleukin-17, Mice, Mice, Inbred C57BL, Myelin-Oligodendrocyte Glycoprotein, Peptide Fragments, Th1 Cells, Thymidine, Tritium
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Animals,Cell Proliferation,drug effects,Central Nervous System,pathology,Cytokines,metabolism,Disease Models, Animal,Encephalomyelitis, Autoimmune, Experimental,chemically induced,Flow Cytometry,methods,Glycoproteins,Interferon-gamma,pharmacology,Interleukin-17,Mice,Mice, Inbred C57BL,Myelin-Oligodendrocyte Glycoprotein,Peptide Fragments,Th1 Cells,Thymidine,Tritium
Animals, Cell Proliferation, drug effects, Central Nervous System, pathology, Cytokines, metabolism, Disease Models, Animal, Encephalomyelitis, Autoimmune, Experimental, chemically induced, Flow Cytometry, methods, Glycoproteins, Interferon-gamma, pharmacology, Interleukin-17, Mice, Mice, Inbred C57BL, Myelin-Oligodendrocyte Glycoprotein, Peptide Fragments, Th1 Cells, Thymidine, Tritium