Early life infection, but not breastfeeding, predicts adult blood telomere lengths in the Philippines: EISENBERG ET AL.

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Abstract

Telomeres are repetitive DNA at chromosomes ends that shorten with age due to cellular replication and oxidative stress. As telomeres shorten, this can eventually place limits on cell replication and contribute to senescence. Infections are common during early development and activate cellular immune responses that involve clonal expansion and oxidative stress. As such, a high infectious disease burden might shorten blood telomere length (BTL) and accelerate the pace of immune senescence.

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Author and article information

Contributors

Dan T.A. Eisenberg: (View ORCID Profile)

Journal

Title: American Journal of Human Biology

Abbreviated Title: Am J Hum Biol

Publisher: Wiley

ISSN: 10420533

Publication date Created: January 2017

Publication date (Print): January 2017

Publication date (Electronic): January 25 2017

Page: e22962

Affiliations

[1 ]Department of Anthropology; University of Washington

[2 ]Center for Studies in Demography and Ecology; University of Washington

[3 ]USC-Office of Population Studies Foundation; University of San Carlos; Cebu City Philippines

[4 ]Department of Nutrition and Dietetics; University of San Carlos; Cebu City Philippines

[5 ]Division of Endocrinology, Metabolism and Molecular Medicine, Department of Medicine; Northwestern University Feinberg School of Medicine; Chicago Illinois

[6 ]Center for Genetic Medicine; Northwestern University Feinberg School of Medicine; Chicago Illinois

[7 ]Department of Anthropology; Northwestern University; Evanston Illinois

[8 ]Institute for Policy Research; Northwestern University; Evanston Illinois