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      Dissecting the genetic predisposition to albuminuria and endothelial dysfunction in a genetic rat model.

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      Journal: Journal of Hypertension
      Publisher: Ovid Technologies (Wolters Kluwer Health)

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          Abstract

          The Munich Wistar Frömter (MWF) rat develops progressive spontaneous albuminuria largely attributable to quantitative trait loci on rat chromosome (RNO)6 and RNO8, respectively. We tested the hypothesis whether quantitative trait loci on these chromosomes are linked to both albuminuria and endothelial dysfunction.

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          PubMed ID:: 23868088
          DOI:: 10.1097/HJH.0b013e3283642384

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