Herpes simplex encephalitis relapse with chorea is associated with autoantibodies to N-Methyl-D-aspartate receptor or dopamine-2 receptor.

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Abstract

Movement disorder relapses after herpes simplex virus 1 (HSV1) encephalitis have been hypothesized to be secondary to postviral autoimmunity. Recently, a proportion of patients with HSV1 encephalitis (HSE) were shown to produce autoantibodies against N-methyl-D-aspartate receptor (NMDAR).

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Journal

Journal ID (iso-abbrev): Mov. Disord.

Title: Movement disorders : official journal of the Movement Disorder Society

ISSN (Electronic): 1531-8257

ISSN (Print): 0885-3185

Publication date (Electronic): Jan 2014

Volume: 29

Issue: 1

Affiliations

[1 ] Neuroimmunology Group, Institute for Neuroscience and Muscle Research, Kids Research Institute at the Children's Hospital at Westmead, University of Sydney, Westmead, Australia; Department of Neurosciences, Royal Children's Hospital, Brisbane, Australia; School of Medicine, University of Queensland, Brisbane, Australia; TY Nelson Department of Neurology and Neurosurgery, Children's Hospital at Westmead, Westmead, Australia.

Article

DOI: 10.1002/mds.25623

PubMed ID: 24115338

SO-VID: e2a1496d-6bcd-4f77-aa55-89563dec9a0c

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Keywords: NMDA receptor,autoantibody,chorea,dopamine-2 receptor,herpes simplex encephalitis

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Keywords: NMDA receptor, autoantibody, chorea, dopamine-2 receptor, herpes simplex encephalitis

Herpes simplex encephalitis relapse with chorea is associated with autoantibodies to N-Methyl-D-aspartate receptor or dopamine-2 receptor.

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