Blog
About

1
views
0
recommends
+1 Recommend
1 collections
    0
    shares
      • Record: found
      • Abstract: found
      • Article: found

      Nonvagally Mediated Bradycardia during Cardiac Tamponade or Severe Hemorrhage

      Read this article at

      ScienceOpenPublisherPubMed
      Bookmark
          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Abstract

          Bradycardia was observed during acute cardiac tamponade and severe hemorrhage in pentobarbital-anesthetized mongrel dogs after bilateral cervical vagotomy, sympathectomy and intravenous atropine. An early and a late bradycardia developed during tamponade. A portion of the early bradycardia was produced by a paradoxical increase in vagal efferent nerve activity and a pacemaker shift; however, after vagotomy, sympathectomy and atropine, a significant (p < 0.02) early and late bradycardia still developed during acute cardiac tamponade and severe hemorrhage. The activation sequence of high and low right atrial electrograms revealed that a pacemaker shift was responsible for the nonvagally mediated bradycardia observed with acute cardiac tamponade and severe hemorrhage. The early and late bradycardias occur with either tamponade or hemorrhage, suggesting that ischemia of the sinoatrial node was the apparent cause of the pacemaker shift and resultant bradycardia.

          Related collections

          Author and article information

          Journal
          CRD
          Cardiology
          10.1159/issn.0008-6312
          Cardiology
          S. Karger AG
          0008-6312
          1421-9751
          1981
          1981
          07 November 2008
          : 68
          : 2
          : 65-79
          Affiliations
          Departments of Anesthesiology, Physiology and Medicine, The Medical College of Wisconsin, and Wood Veterans Administration Medical Center, Wood, Milwaukee, Wisc., USA
          Article
          173266 Cardiology 1981;68:65–79
          10.1159/000173266
          7273047
          © 1981 S. Karger AG, Basel

          Copyright: All rights reserved. No part of this publication may be translated into other languages, reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, microcopying, or by any information storage and retrieval system, without permission in writing from the publisher. Drug Dosage: The authors and the publisher have exerted every effort to ensure that drug selection and dosage set forth in this text are in accord with current recommendations and practice at the time of publication. However, in view of ongoing research, changes in government regulations, and the constant flow of information relating to drug therapy and drug reactions, the reader is urged to check the package insert for each drug for any changes in indications and dosage and for added warnings and precautions. This is particularly important when the recommended agent is a new and/or infrequently employed drug. Disclaimer: The statements, opinions and data contained in this publication are solely those of the individual authors and contributors and not of the publishers and the editor(s). The appearance of advertisements or/and product references in the publication is not a warranty, endorsement, or approval of the products or services advertised or of their effectiveness, quality or safety. The publisher and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements.

          Page count
          Pages: 15
          Categories
          Original Paper

          Comments

          Comment on this article