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Smad3 mutant mice develop metastatic colorectal cancer.
1998-09-18
Adenocarcinoma, genetics, mortality, secondary, Adenomatous Polyposis Coli Protein, Alleles, Animals, Cell Division, Cloning, Molecular, Colorectal Neoplasms, Cytoskeletal Proteins, DNA-Binding Proteins, Female, Gene Expression Regulation, Developmental, Gene Expression Regulation, Neoplastic, Genes, Recessive, Genes, Reporter, Heterozygote, Hybridization, Genetic, Lac Operon, Lymph Nodes, pathology, Male, Mice, Mice, Inbred C57BL, Mice, Mutant Strains, Signal Transduction, Smad3 Protein, Survival Analysis, Trans-Activators, Xenopus
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Adenocarcinoma,genetics,mortality,secondary,Adenomatous Polyposis Coli Protein,Alleles,Animals,Cell Division,Cloning, Molecular,Colorectal Neoplasms,Cytoskeletal Proteins,DNA-Binding Proteins,Female,Gene Expression Regulation, Developmental,Gene Expression Regulation, Neoplastic,Genes, Recessive,Genes, Reporter,Heterozygote,Hybridization, Genetic,Lac Operon,Lymph Nodes,pathology,Male,Mice,Mice, Inbred C57BL,Mice, Mutant Strains,Signal Transduction,Smad3 Protein,Survival Analysis,Trans-Activators,Xenopus
Adenocarcinoma, genetics, mortality, secondary, Adenomatous Polyposis Coli Protein, Alleles, Animals, Cell Division, Cloning, Molecular, Colorectal Neoplasms, Cytoskeletal Proteins, DNA-Binding Proteins, Female, Gene Expression Regulation, Developmental, Gene Expression Regulation, Neoplastic, Genes, Recessive, Genes, Reporter, Heterozygote, Hybridization, Genetic, Lac Operon, Lymph Nodes, pathology, Male, Mice, Mice, Inbred C57BL, Mice, Mutant Strains, Signal Transduction, Smad3 Protein, Survival Analysis, Trans-Activators, Xenopus