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      Turnover of type III collagen reflects disease severity and is associated with progression and microinflammation in patients with IgA nephropathy.

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          Abstract

          Renal fibrosis is the hallmark of progression to end-stage kidney disease. Non-invasive biomarkers of renal fibrosis could serve as specific end points in clinical trials and improve monitoring and stratification of patients with chronic kidney disease (CKD). To address this, we measured markers of collagen type III turnover in urine and serum of IgA nephropathy (IgAN) patients.

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          Author and article information

          Journal
          Nephrol. Dial. Transplant.
          Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
          Oxford University Press (OUP)
          1460-2385
          0931-0509
          Mar 2016
          : 31
          : 3
          Affiliations
          [1 ] Nordic Bioscience, Herlev, Denmark.
          [2 ] Nephrology and Immunology, RWTH University of Aachen, Aachen, NRW, Germany Institute of Pathology, RWTH University of Aachen, Aachen, NRW, Germany Institute of Molecular Biomedicine, Comenius University, Bratislava, Slovakia.
          [3 ] Nephrology and Immunology, RWTH University of Aachen, Aachen, NRW, Germany Institute of Pathology, RWTH University of Aachen, Aachen, NRW, Germany Department of Nephrology, University Hospital of Patras, Patras, Greece.
          [4 ] Nephrology and Immunology, RWTH University of Aachen, Aachen, NRW, Germany.
          Article
          gfv301
          10.1093/ndt/gfv301
          26311218
          eaffad94-558e-4bd0-ac11-3be167ee52f8
          History

          IgAN,biomarkers,collagen,fibrosis,turnover
          IgAN, biomarkers, collagen, fibrosis, turnover

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