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      Paradoxical activation of endothelial nitric oxide synthase by NADPH oxidase.

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          Abstract

          Increased formation of reactive oxygen species (ROS) has been identified as a causative factor in endothelial dysfunction by reducing NO bioavailability and uncoupling endothelial nitric oxide synthase (eNOS). However, the specific contribution of ROS to endothelial function is not well understood.

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          Author and article information

          Journal
          Arterioscler Thromb Vasc Biol
          Arteriosclerosis, thrombosis, and vascular biology
          Ovid Technologies (Wolters Kluwer Health)
          1524-4636
          1079-5642
          Sep 2008
          : 28
          : 9
          Affiliations
          [1 ] Department of Pharmacology, Vascular Biology Center, Medical College of Georgia, Augusta, GA 30912-2500, USA.
          Article
          ATVBAHA.108.168278 NIHMS126008
          10.1161/ATVBAHA.108.168278
          2771553
          18556569
          ef3a0543-004e-4d0f-924f-66a0149dbfeb
          History

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