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      The Role of the Autonomic Nerves in the Control of Nasal Circulation

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          Abstract

          Patients suffering from allergic or vasomotor rhinitis usually show nasal mucosal hyperaemia, engorgement, hyperrhinorrhoea and obstruction of the nasal airway. The nasal mucosa is drained by two venous systems which are anatomically and functionally separate. The nasal mucosa receives tone discharges from the sympathetic nerves but not from the parasympathetic nerves. Sympathetic nerve stimulation causes constriction of the resistance vessels via the α -adrenergic mechanism and constriction of the capacitance vessels via the α -adrenergic mechanism and some non-adrenergic and non-cholinergic mechanism; the capacitance vessels are under more prominent sympathetic influence than the resistance vessels. Parasympathetic nerve stimulation causes non-cholinergic dilatation of both resistance and capacitance vessels; dilatation is more pronounced in the posterior venous system. Simultaneous optimal stimulation of the autonomic nerves resulted in vasoconstriction, especially of the capacitance vessels. Hence, nasal congestion may be related more to a withdrawal of sympathetic discharge than to an overactivity of the parasympathetic nerves.

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          Author and article information

          Journal
          NSG
          Neurosignals
          10.1159/issn.1424-862X
          Neurosignals
          S. Karger AG
          978-3-8055-6264-5
          978-3-8055-8877-5
          1424-862X
          1424-8638
          1995
          1995
          15 December 1995
          : 4
          : 3
          : 179-185
          Affiliations
          Department of Physiology, Faculty of Medicine, University of Hong Kong, Hong Kong
          Article
          109439 Neurosignals 1995;4:179–185
          10.1159/000109439
          © 1995 S. Karger AG, Basel

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          Page count
          Pages: 7
          Categories
          Cardiovascular Sciences

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