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      Autotaxin interacts with lipoprotein(a) and oxidized phospholipids in predicting the risk of calcific aortic valve stenosis in patients with coronary artery disease.

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          Abstract

          Studies have shown that lipoprotein(a) [Lp(a)], an important carrier of oxidized phospholipids, is causally related to calcific aortic valve stenosis (CAVS). Recently, we found that Lp(a) mediates the development of CAVS through autotaxin (ATX).

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          Author and article information

          Journal
          J. Intern. Med.
          Journal of internal medicine
          Wiley
          1365-2796
          0954-6820
          Nov 2016
          : 280
          : 5
          Affiliations
          [1 ] Laboratory of Cardiovascular Pathobiology Quebec Heart and Lung Institute/Research Center, Department of Surgery, Quebec, Canada.
          [2 ] Department of Medicine, Laval University, Quebec, Canada.
          [3 ] Statistical Consulting Service Unit at the Quebec Heart and Lung Institute/Research Center, Laval University, Quebec, Canada.
          [4 ] University of California San Diego, La Jolla, CA, USA.
          [5 ] Department of Molecular Medicine, Laval University, Quebec, Canada.
          [6 ] Laboratory of Cardiovascular Pathobiology Quebec Heart and Lung Institute/Research Center, Department of Surgery, Quebec, Canada. patrick.mathieu@chg.ulaval.ca.
          Article
          10.1111/joim.12519
          27237700

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