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      Treatment for Anhedonia: A Neuroscience Driven Approach : 2015 ADAA Scientific Research Symposium: Treatment for Anhedonia

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          The positive and negative affect schedule (PANAS): construct validity, measurement properties and normative data in a large non-clinical sample.

          To evaluate the reliability and validity of the PANAS (Watson, Clark, & Tellegen, 1988b) and provide normative data. Cross-sectional and correlational. The PANAS was administered to a non-clinical sample, broadly representative of the general adult UK population (N = 1,003). Competing models of the latent structure of the PANAS were evaluated using confirmatory factor analysis. Regression and correlational analysis were used to determine the influence of demographic variables on PANAS scores as well as the relationship between the PANAS with measures of depression and anxiety (the HADS and the DASS). The best-fitting model (robust comparative fit index = .94) of the latent structure of the PANAS consisted of two correlated factors corresponding to the PA and NA scales, and permitted correlated error between items drawn from the same mood subcategories (Zevon & Tellegen, 1982). Demographic variables had only very modest influences on PANAS scores and the PANAS exhibited measurement invariance across demographic subgroups. The reliability of the PANAS was high, and the pattern of relationships between the PANAS and the DASS and HADS were consistent with tripartite theory. The PANAS is a reliable and valid measure of the constructs it was intended to assess, although the hypothesis of complete independence between PA and NA must be rejected. The utility of this measure is enhanced by the provision of large-scale normative data. Copyright 2004 The British Psychological Society
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            Reconsidering anhedonia in depression: lessons from translational neuroscience.

            Anhedonia is a core symptom of major depressive disorder (MDD), the neurobiological mechanisms of which remain poorly understood. Despite decades of speculation regarding the role of dopamine (DA) in anhedonic symptoms, empirical evidence has remained elusive, with frequent reports of contradictory findings. In the present review, we argue that this has resulted from an underspecified definition of anhedonia, which has failed to dissociate between consummatory and motivational aspects of reward behavior. Given substantial preclinical evidence that DA is involved primarily in motivational aspects of reward, we suggest that a refined definition of anhedonia that distinguishes between deficits in pleasure and motivation is essential for the purposes of identifying its neurobiological substrates. Moreover, bridging the gap between preclinical and clinical models of anhedonia may require moving away from the conceptualization of anhedonia as a steady-state, mood-like phenomena. Consequently, we introduce the term "decisional anhedonia" to address the influence of anhedonia on reward decision-making. These proposed modifications to the theoretical definition of anhedonia have implications for research, assessment and treatment of MDD. Copyright © 2010 Elsevier Ltd. All rights reserved.
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              Brain Systems that Mediate both Emotion and Cognition

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                Author and article information

                Journal
                Depression and Anxiety
                Depress Anxiety
                Wiley
                10914269
                October 2016
                October 2016
                October 03 2016
                : 33
                : 10
                : 927-938
                Affiliations
                [1 ]Department of Psychology; University of California; Los Angeles California
                [2 ]Department of Psychology; Southern Methodist University; Dallas Texas
                Article
                10.1002/da.22490
                ffa829fb-9de2-4634-ad1e-d3815c09ba77
                © 2016
                Product
                Self URI (article page): http://doi.wiley.com/10.1002/da.22490

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