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      Call for Papers: Preclinical Investigations of Nutrigenetic/Nutrigenomic Targets

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      About Lifestyle Genomics: 2.6 Impact Factor I 3.7 CiteScore I 0.539 Scimago Journal & Country Rank (SJR)

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      Relationships between dietary intake and body composition according to gross motor functional ability in preschool-aged children with cerebral palsy.

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          We aimed to determine the relationships between energy intake, macronutrient intake and body composition in preschool-aged children with cerebral palsy (CP) according to gross motor functional ability in comparison with typically developing children (TDC).

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          Dietary risk factors for development of childhood obesity.

          Rosa L Moreno, Gerardo Rodriguez-Araujo (2007)
          Controversial information exists on the contribution of several dietary factors for overweight development in childhood, but there is no doubt that obesity prevalence is increasing. We review the most up-to-date information in order to clarify the evidence-based dietary aspects influencing obesity development in children and adolescents. Longitudinal studies are the preferred method for analysing the relationship between dietary factors and obesity development. With the exception of infants, there are no conclusive associations between energy intake or diet composition and later overweight development in children. Among formula or mixed-fed infants, the increase in energy intake has been associated with an increased risk of being overweight during childhood. Breastfeeding seems to be a protective factor for later obesity development. In terms of food intake, longitudinal studies have only found a clear and positive association between obesity development and sugar-sweetened beverage consumption; this is not the case with snacking, fast food or food portion sizes. Cross-sectional studies have found correlations between being overweight in childhood and buying lunch at school, eating supper while watching television or without family supervision, consuming less energy at breakfast or more at dinner, and missing breakfast. Results from longitudinal studies must be taken into account in order to design preventive strategies to counteract the increased prevalence of obesity and its consequences in children. Lack of breastfeeding, high early energy intake and high intake of sugar-sweetened beverages seem to be the main dietary factors contributing to obesity development.
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            Dysphagia in children with severe generalized cerebral palsy and intellectual disability.

            Rebekka Veugelers, Heleen M Evenhuis, Corine Penning … (2008)
            This study assessed the clinical indicators and severity of dysphagia in a representative sample of children with severe generalized cerebral palsy and intellectual disability. A total of 166 children (85 males, 81 females) with Gross Motor Function Classification System Level IV or V and IQ<55 were recruited from 54 daycare centres. Mean age was 9 years 4 months (range 2 y 1 mo-19 y 1 mo). Clinically apparent presence and severity of dysphagia were assessed with a standardized mealtime observation, the Dysphagia Disorders Survey (DDS), and a dysphagia severity scale. Additional measures were parental report on feeding problems and mealtime duration. Of all 166 participating children, 1% had no dysphagia, 8% mild dysphagia, 76% moderate to severe dysphagia, and 15% profound dysphagia (receiving nil by mouth), resulting in a prevalence of dysphagia of 99%. Dysphagia was positively related to severity of motor impairment, and, surprisingly, to a higher weight for height. Low frequency of parent-reported feeding problems indicated that actual severity of dysphagia tended to be underestimated by parents. Proactive identification of dysphagia is warranted in this population, and feasible using a structured mealtime observation. Children with problems in the pharyngeal and esophageal phases, apparent on the DDS, should be referred for appropriate clinical evaluation of swallowing function.
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              Growth and nutrition disorders in children with cerebral palsy.

              Michelle Kuperminc, Richard Stevenson (2008)
              Growth and nutrition disorders are common secondary health conditions in children with cerebral palsy (CP). Poor growth and malnutrition in CP merit study because of their impact on health, including psychological and physiological function, healthcare utilization, societal participation, motor function, and survival. Understanding the etiology of poor growth has led to a variety of interventions to improve growth. One of the major causes of poor growth, malnutrition, is the best-studied contributor to poor growth; scientific evidence regarding malnutrition has contributed to improvements in clinical management and, in turn, survival over the last 20 years. Increased recognition and understanding of neurological, endocrinological, and environmental factors have begun to shape care for children with CP, as well. The investigation of these factors relies on advances made in the assessment methods available to address the challenges inherent in measuring growth in children with CP. Descriptive growth charts and norms of body composition provide information that may help clinicians to interpret growth and intervene to improve growth and nutrition in children with CP. Linking growth to measures of health will be necessary to develop growth standards for children with CP in order to optimize health and well-being. (c) 2008 Wiley-Liss, Inc.
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                Author and article information

                Journal
                Journal ID (iso-abbrev): Ann. Nutr. Metab.
                Title: Annals of nutrition & metabolism
                ISSN (Electronic): 1421-9697
                ISSN (Print): 0250-6807
                Publication date (Electronic): 2012
                Volume: 61
                Issue: 4
                Affiliations
                [1 ] Children's Nutrition Research Centre, University of Queensland, Royal Children's Hospital, Brisbane, Qld., Australia. j.walker3@uq.edu.au
                Article
                Publisher Item ID: 000342557
                DOI: 10.1159/000342557
                PubMed ID: 23208164
                SO-VID: 9df77647-f0c3-4529-a765-4cf09b75ac95
                Copyright © Copyright © 2012 S. Karger AG, Basel.
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