1
views
0
recommends
+1 Recommend
0 collections
    0
    shares
      • Record: found
      • Abstract: found
      • Article: found
      Is Open Access

      Noodles Made from High Amylose Wheat Flour Attenuate Postprandial Glycaemia in Healthy Adults

      research-article

      Read this article at

      Bookmark
          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Abstract

          Previous research has not considered the effect of high amylose wheat noodles on postprandial glycaemia. The aim of the study is to investigate the effect of consumption of high amylose noodles on postprandial glycaemia over 2-h periods by monitoring changes in blood glucose concentration and calculating the total area under the blood glucose concentration curve. Twelve healthy young adults were recruited to a repeated measure randomised, single-blinded crossover trial to compare the effect of consuming noodles (180 g) containing 15%, 20% and 45% amylose on postprandial glycaemia. Fasting blood glucose concentrations were taken via finger-prick blood samples. Postprandial blood glucose concentrations were taken at 15, 30, 45, 60, 90 and 120 min. Subjects consuming high amylose noodles made with flour containing 45% amylose had significantly lower blood glucose concentration at 15, 30 and 45 min (5.5 ± 0.11, 6.1 ± 0.11 and 5.6 ± 0.11 mmol/L; p = 0.01) compared to subjects consuming low amylose noodles with 15% amylose (5.8 ± 0.12, 6.6 ± 0.12 and 5.9 ± 0.12 mmol/L). The total area under the blood glucose concentration curve after consumption of high amylose noodles with 45% amylose was 640.4 ± 9.49 mmol/L/min, 3.4% lower than consumption of low amylose noodles with 15% amylose (662.9 ± 9.49 mmol/L/min), p = 0.021. Noodles made from high amylose wheat flour attenuate postprandial glycaemia in healthy young adults, as characterised by the significantly lower blood glucose concentration and a 3.4% reduction in glycaemic response.

          Related collections

          Most cited references50

          • Record: found
          • Abstract: not found
          • Article: not found

          The molecular structures of starch components and their contribution to the architecture of starch granules: A comprehensive review

            Bookmark
            • Record: found
            • Abstract: found
            • Article: found
            Is Open Access

            Impact of postprandial glycaemia on health and prevention of disease

            Postprandial glucose, together with related hyperinsulinemia and lipidaemia, has been implicated in the development of chronic metabolic diseases like obesity, type 2 diabetes mellitus (T2DM) and cardiovascular disease (CVD). In this review, available evidence is discussed on postprandial glucose in relation to body weight control, the development of oxidative stress, T2DM, and CVD and in maintaining optimal exercise and cognitive performance. There is mechanistic evidence linking postprandial glycaemia or glycaemic variability to the development of these conditions or in the impairment in cognitive and exercise performance. Nevertheless, postprandial glycaemia is interrelated with many other (risk) factors as well as to fasting glucose. In many studies, meal-related glycaemic response is not sufficiently characterized, or the methodology with respect to the description of food or meal composition, or the duration of the measurement of postprandial glycaemia is limited. It is evident that more randomized controlled dietary intervention trials using effective low vs. high glucose response diets are necessary in order to draw more definite conclusions on the role of postprandial glycaemia in relation to health and disease. Also of importance is the evaluation of the potential role of the time course of postprandial glycaemia.
              Bookmark
              • Record: found
              • Abstract: found
              • Article: not found

              Molecular disassembly of starch granules during gelatinization and its effect on starch digestibility: a review.

              Starch is the most important glycemic carbohydrate in foods. The relationship between the rate and extent of starch digestion to produce glucose for absorption into the bloodstream and risk factors for diet-related diseases is of considerable nutritional interest. Native starch is attacked slowly by enzymes, but after hydrothermal processing its susceptibility to enzymatic breakdown is greatly increased. Most starch consumed by humans has undergone some form of processing or cooking, which causes native starch granules to gelatinize, followed by retrogradation on cooling. The extent of gelatinization and retrogradation are major determinants of the susceptibility of starch to enzymatic digestion and its functional properties for food processing. The type and extent of changes that occur in starch as a result of gelatinization, pasting and retrogradation are determined by the type of the starch, processing and storage conditions. A mechanistic understanding of the molecular disassembly of starch granules during gelatinization is critical to explaining the effects of processing or cooking on starch digestibility. This review focuses on the molecular disassembly of starch granules during starch gelatinization over a wide range of water levels, and its consequential effect on in vitro starch digestibility and in vivo glycemic index.
                Bookmark

                Author and article information

                Journal
                Nutrients
                Nutrients
                nutrients
                Nutrients
                MDPI
                2072-6643
                22 July 2020
                August 2020
                : 12
                : 8
                : 2171
                Affiliations
                [1 ]School Public Health, Curtin University, Perth 6845, Western Australia, Australia; kimang03@ 123456yahoo.com.au (K.A.); carla.bourgy@ 123456blackmores.com.au (C.B.); h.fenton@ 123456curtin.edu.au (H.F.); wendy.hunt@ 123456aegic.org.au (W.H.)
                [2 ]School of Molecular and Life Sciences, Curtin University, Perth 6845, Western Australia, Australia
                [3 ]Commonwealth Scientific and Industrial Research Organisation, Agriculture and Food, Canberra 2601, ACT, Australia; a.regina@ 123456irri.org (A.R.); marcus.newberry@ 123456csiro.au (M.N.)
                [4 ]Department of Primary Industries and Regional Development, South Perth 6151, Western Australia; Australia; dean.diepeveen@ 123456dpird.wa.gov.au
                [5 ]College of Science, Health, Engineering and Education, Murdoch University, Murdoch 6150, Western Australia, Australia
                [6 ]Department Agricultural and Forestry Sciences, University of Tuscia, 01100 Viterbo, Italy; lafiandr@ 123456unitus.it
                [7 ]Grains and Legumes Nutrition Council, North Ryde 2113, Australia; sarag@ 123456glnc.org.au
                [8 ]Australian Export Grains Innovation Centre, South Perth 6151, Western Australia, Australia
                Author notes
                Author information
                https://orcid.org/0000-0002-1702-0038
                https://orcid.org/0000-0002-4286-8284
                https://orcid.org/0000-0003-4029-5977
                Article
                nutrients-12-02171
                10.3390/nu12082171
                7468775
                32707905
                2029202d-ec4f-4a3b-a590-99b9a0f53287
                © 2020 by the authors.

                Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license ( http://creativecommons.org/licenses/by/4.0/).

                History
                : 19 June 2020
                : 21 July 2020
                Categories
                Article

                Nutrition & Dietetics
                blood glucose,glycaemic response,resistant starch
                Nutrition & Dietetics
                blood glucose, glycaemic response, resistant starch

                Comments

                Comment on this article