Long non-coding MIAT mediates high glucose-induced renal tubular epithelial injury.

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Abstract

Long non-coding RNAs (lncRNAs) constitute a novel class of non-coding RNAs that take part in occurrence and development of diabetes complication via regulating gene expression. However, litter is known about lncRNAs in the setting of diabetes induced nephropathy. The aim of this study was to examine whether lncRNA-myocardial infarction-associated transcript (MIAT) is involved in diabetes induced renal tubules injury.

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Author and article information

Journal

Journal ID (iso-abbrev): Biochem. Biophys. Res. Commun.

Title: Biochemical and biophysical research communications

Publisher: Elsevier BV

ISSN (Electronic): 1090-2104

ISSN (Print): 0006-291X

Publication date (Electronic): Dec 25 2015

Volume: 468

Issue: 4

Affiliations

[1 ] Department of Nephrology, The First Affiliated Hospital of Soochow University, Suzhou, 215006, China.

[2 ] Department of Nephrology, The First Affiliated Hospital of Soochow University, Suzhou, 215006, China. Electronic address: guoyuan_l@163.com.

[3 ] Department of Endocrinology, The First Affiliated Hospital of Soochow University, Suzhou, 215006, China.

Article

Publisher Item ID: S0006-291X(15)30893-7

DOI: 10.1016/j.bbrc.2015.11.023

PubMed ID: 26551455

SO-VID: 3b528862-ed7f-4221-b8e1-0f731a13e5cf

History

Keywords: Serum creatinine,Cell viability,HK-2,Nrf2

Data availability:

Keywords: Serum creatinine, Cell viability, HK-2, Nrf2

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