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      Resolution of manic symptoms following unilateral adrenalectomy in patient with primary bilateral macronodular adrenal hyperplasia

      case-report

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          There is no author summary for this article yet. Authors can add summaries to their articles on ScienceOpen to make them more accessible to a non-specialist audience.

          Abstract

          Primary bilateral macronodular adrenal hyperplasia (PBMAH) is often associated with symptoms of cortisol excess, which may include neuropsychological symptoms. We report a patient with PBMAH who presented with manic symptoms that resolved following unilateral adrenalectomy.

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          Neuropsychiatric disorders in Cushing's syndrome

          Rosario Pivonello, Chiara Simeoli, Maria Cristina De Martino … (2015)
          Endogenous Cushing's syndrome (CS), a rare endocrine disorder characterized by cortisol hypersecretion, is associated with psychiatric and neurocognitive disorders. Major depression, mania, anxiety, and neurocognitive impairment are the most important clinical abnormalities. Moreover, patients most often complain of impairment in quality of life, interference with family life, social, and work performance. Surprisingly, after hypercortisolism resolution, despite the improvement of the overall prevalence of psychiatric and neurocognitive disorders, the brain volume loss at least partially persists and it should be noted that some patients may still display depression, anxiety, panic disorders, and neurocognitive impairment. This brief review aimed at describing the prevalence of psychiatric and neurocognitive disorders and their characterization both during the active and remission phases of CS. The last section of this review is dedicated to quality of life, impaired during active CS and only partially resolved after resolution of hypercortisolism.
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            Bilateral Adrenal Hyperplasia: Pathogenesis and Treatment.

            Benjamin Chevalier, Marie-Christine Vantyghem, Stéphanie Espiard (2021)
            Bilateral adrenal hyperplasia is a rare cause of Cushing's syndrome. Micronodular adrenal hyperplasia, including the primary pigmented micronodular adrenal dysplasia (PPNAD) and the isolated micronodular adrenal hyperplasia (iMAD), can be distinguished from the primary bilateral macronodular adrenal hyperplasia (PBMAH) according to the size of the nodules. They both lead to overt or subclinical CS. In the latter case, PPNAD is usually diagnosed after a systematic screening in patients presenting with Carney complex, while for PBMAH, the diagnosis is often incidental on imaging. Identification of causal genes and genetic counseling also help in the diagnoses. This review discusses the last decades' findings on genetic and molecular causes of bilateral adrenal hyperplasia, including the several mechanisms altering the PKA pathway, the recent discovery of ARMC5, and the role of the adrenal paracrine regulation. Finally, the treatment of bilateral adrenal hyperplasia will be discussed, focusing on current data on unilateral adrenalectomy.
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              ACTH-independent macronodular adrenal hyperplasia

              A. LaCroix (2024)
              Article 0 comments Cited 1 times – based on 0 reviews     Review now
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                Author and article information

                Contributors
                Benjamin H. Rosenfeld
                Benjamin T. Ristau
                Journal
                Journal ID (nlm-ta): Urol Case Rep
                Journal ID (iso-abbrev): Urol Case Rep
                Title: Urology Case Reports
                Publisher: Elsevier
                ISSN (Electronic): 2214-4420
                Publication date PMC-release: 10 November 2022
                Publication date Collection: November 2022
                Publication date (Electronic): 10 November 2022
                Volume: 45
                Electronic Location Identifier: 102277
                Affiliations
                [1]UConn Health, United States
                Author notes
                [∗ ]Corresponding author. benristaumd@ 123456gmail.com
                Article
                Publisher Item ID: S2214-4420(22)00289-3 Publisher ID: 102277
                DOI: 10.1016/j.eucr.2022.102277
                PMC ID: 9668685
                PubMed ID: 36406008
                SO-VID: 751e377f-de77-4d36-9c81-626c02df0d06
                Copyright © © 2022 The Author(s)
                License:

                This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

                History
                Date received : 27 September 2022
                Date accepted : 2 November 2022
                Categories
                Subject: Oncology

                Data availability:

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