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Quercetin, a bioflavonoid, attenuates mechanical allodynia in a rat model of cancer-induced bone pain via suppressing the PI3Kγ/Akt signaling pathway
Author(s):
D. Liu
,
Y. Zhou
,
Y. Tian
,
D. Ye
Publication date
Created:
March 2018
Publication date
(Print):
March 2018
Journal:
The Journal of Pain
Publisher:
Elsevier BV
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Neuronal Signaling
Author and article information
Journal
Title:
The Journal of Pain
Abbreviated Title:
The Journal of Pain
Publisher:
Elsevier BV
ISSN (Print):
15265900
Publication date Created:
March 2018
Publication date (Print):
March 2018
Volume
: 19
Issue
: 3
Page
: S77
Article
DOI:
10.1016/j.jpain.2017.12.181
SO-VID:
a9445b54-6af1-4401-af7b-bef47a2ed8ae
Copyright ©
© 2018
License:
https://www.elsevier.com/tdm/userlicense/1.0/
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Gγ-like (ggl) domains: new frontiers in g-protein signaling and β-propeller scaffolding22Abbreviations: DEP, dishevelled/EGL-10/pleckstrin-related domain; DH, dbl-homology domain; GAP, guanosine triphosphatase-activating protein; GEF, guanine nucleotide exchange factor; GGL, G-gamma-like; GIRK, G-protein-gated inwardly rectifying potassium channel; GPCR, G-protein-coupled receptor; G protein, guanine nucleotide binding protein; GTPase, guanosine triphosphatase; mAChR, muscarinic acetylcholine receptor; MAPK, mitogen-activated protein kinase; PDE, phosphodiesterase; PDZ, PSD-95/Discs-large/ZO-1 related domain; PH, pleckstrin-homology domain; PI3Kγ, gamma isoform of phosphoinositide 3-kinase; PLC-β, beta isoform of phospholipase C; PTB, phosphotyrosine-binding domain; RACK1, receptor for activated C kinase type-1; RBD, Ras-binding domain; RGS, regulators of G-protein signaling; and SAPK, stress-activated protein kinase.
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