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      Spatial Growth of Real-world Networks

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          Abstract

          Many real-world networks have properties of small-world networks, with clustered local neighborhoods and low average-shortest path (ASP). They may also show a scale-free degree distribution, which can be generated by growth and preferential attachment to highly connected nodes, or hubs. However, many real-world networks consist of multiple, inter-connected clusters not normally seen in systems grown by preferential attachment, and there also exist real-world networks with a scale-free degree distribution that do not contain highly connected hubs. We describe spatial growth mechanisms, not using preferential attachment, that address both aspects.

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          Emergence of scaling in random networks

          Systems as diverse as genetic networks or the world wide web are best described as networks with complex topology. A common property of many large networks is that the vertex connectivities follow a scale-free power-law distribution. This feature is found to be a consequence of the two generic mechanisms that networks expand continuously by the addition of new vertices, and new vertices attach preferentially to already well connected sites. A model based on these two ingredients reproduces the observed stationary scale-free distributions, indicating that the development of large networks is governed by robust self-organizing phenomena that go beyond the particulars of the individual systems.
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            Statistical mechanics of complex networks

            Complex networks describe a wide range of systems in nature and society, much quoted examples including the cell, a network of chemicals linked by chemical reactions, or the Internet, a network of routers and computers connected by physical links. While traditionally these systems were modeled as random graphs, it is increasingly recognized that the topology and evolution of real networks is governed by robust organizing principles. Here we review the recent advances in the field of complex networks, focusing on the statistical mechanics of network topology and dynamics. After reviewing the empirical data that motivated the recent interest in networks, we discuss the main models and analytical tools, covering random graphs, small-world and scale-free networks, as well as the interplay between topology and the network's robustness against failures and attacks.
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              Lethality and centrality in protein networks

              In this paper we present the first mathematical analysis of the protein interaction network found in the yeast, S. cerevisiae. We show that, (a) the identified protein network display a characteristic scale-free topology that demonstrate striking similarity to the inherent organization of metabolic networks in particular, and to that of robust and error-tolerant networks in general. (b) the likelihood that deletion of an individual gene product will prove lethal for the yeast cell clearly correlates with the number of interactions the protein has, meaning that highly-connected proteins are more likely to prove essential than proteins with low number of links to other proteins. These results suggest that a scale-free architecture is a generic property of cellular networks attributable to universal self-organizing principles of robust and error-tolerant networks and that will likely to represent a generic topology for protein-protein interactions.
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                Author and article information

                Journal
                01 December 2003
                Article
                10.1103/PhysRevE.69.036103
                cond-mat/0312037
                b9d30884-aba5-4043-8673-b0a93439acae
                History
                Custom metadata
                Phys. Rev. E 69:036103 (2004)
                6 pages, 4 figures
                cond-mat.stat-mech

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