PI3K Catalytic Isoform Alteration Promotes the LIMK1-related Metastasis Through the PAK1 or ROCK1/2 Activation in Cigarette Smoke-exposed Ovarian Cancer Cells.

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Abstract

To investigate the molecular mechanisms by which long-term exposure to cigarette smoke extract (CSE) contributes to ovarian cancer metastasis.

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Journal ID (iso-abbrev): Anticancer Res.

Title: Anticancer research

Publisher: International Institute of Anticancer Research

ISSN (Electronic): 1791-7530

ISSN (Print): 0250-7005

Publication date (Electronic): April 2017

Volume: 37

Issue: 4

Affiliations

[1 ] Department of Biochemistry, Kosin University College of Medicine, Busan, Republic of Korea.

[2 ] Department of Anatomy, Inje University College of Medicine, Busan, Republic of Korea kimdj@inje.ac.kr.

Article

Publisher Item ID: 37/4/1805

DOI: 10.21873/anticanres.11515

PubMed ID: 28373445

SO-VID: 978223bc-14e0-4ba8-a048-db9f2d851667

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Keywords: rho-associated kinases 1/2,Phosphoinositide 3-kinase,cigarette smoke extract,p21 protein-activated kinase 1,ovarian cancer,epithelial-mesenchymal transition,LIM kinase 1

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Keywords: rho-associated kinases 1/2, Phosphoinositide 3-kinase, cigarette smoke extract, p21 protein-activated kinase 1, ovarian cancer, epithelial-mesenchymal transition, LIM kinase 1

PI3K Catalytic Isoform Alteration Promotes the LIMK1-related Metastasis Through the PAK1 or ROCK1/2 Activation in Cigarette Smoke-exposed Ovarian Cancer Cells.

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