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      Effects of Extracorporeal Shock Wave Lithotripsy on Renal Function in Patients with Kidney Stone Disease

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          Abstract

          The effects of extracorporeal shock wave lithotripsy on glomerular and tubular renal functions were determined by serum β<sub>2</sub>-microglobulin (Sβ<sub>2</sub>m) and urinary β<sub>2</sub>-microglobulin (Uβ<sub>2</sub>m) estimations in patients with nephrolithiasis. Unilateral treatment was performed in all patients. Urinary and serum creatinine levels were determined according to the method of Yatzidis. Sβ<sub>2</sub>m and Uβ<sub>2</sub>m were measured by radioimmunoassay the day before ESWL, on the day of ESWL, and then 1, 2, 5, 7, 8, 9, 14, and 28 days after treatment. Creatinine clearance, hourly urinary β<sub>2</sub>m excretion (Uβ<sub>2</sub>m/h), and tubular reabsorption of β<sub>2</sub>m (TRβ<sub>2</sub>m) were calculated. After lithotripsy, significant increases in Uβ<sub>2</sub>m, Uβ<sub>2</sub>m/h, and TRβ<sub>2</sub>m were found (p < 0.001), whereas Sβ<sub>2</sub>m, serum creatinine, and creatinine clearance values remained unchanged. Uβ<sub>2</sub>m, Uβ<sub>2</sub>m/h, and TRβ<sub>2</sub>m reached their pretreatment values within 7–9 days after ESWL. We concluded that ESWL does not affect the glomerular filtration rate; however, it leads to a transient proximal tubular dysfunction.

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          Most cited references 2

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          Biological effects of shock waves: Kidney damage by shock waves in dogs—Dose dependence

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            Increased concentration of circulating acid glycosaminoglycans in chronic lymphocytic leukaemia and essential thrombocythaemia

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              Author and article information

              Journal
              NEF
              Nephron
              10.1159/issn.1660-8151
              Nephron
              S. Karger AG
              1660-8151
              2235-3186
              1998
              June 1998
              27 May 1998
              : 79
              : 2
              : 162-166
              Affiliations
              Department of Hypertension, Karol Marcinkowski School of Medical Sciences, Poznań, Poland
              Article
              45019 Nephron 1998;79:162–166
              10.1159/000045019
              9647495
              © 1998 S. Karger AG, Basel

              Copyright: All rights reserved. No part of this publication may be translated into other languages, reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, microcopying, or by any information storage and retrieval system, without permission in writing from the publisher. Drug Dosage: The authors and the publisher have exerted every effort to ensure that drug selection and dosage set forth in this text are in accord with current recommendations and practice at the time of publication. However, in view of ongoing research, changes in government regulations, and the constant flow of information relating to drug therapy and drug reactions, the reader is urged to check the package insert for each drug for any changes in indications and dosage and for added warnings and precautions. This is particularly important when the recommended agent is a new and/or infrequently employed drug. Disclaimer: The statements, opinions and data contained in this publication are solely those of the individual authors and contributors and not of the publishers and the editor(s). The appearance of advertisements or/and product references in the publication is not a warranty, endorsement, or approval of the products or services advertised or of their effectiveness, quality or safety. The publisher and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements.

              Page count
              Tables: 2, References: 31, Pages: 5
              Product
              Self URI (application/pdf): https://www.karger.com/Article/Pdf/45019
              Categories
              Original Paper

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