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      Value Assessment of Health Losses Caused by PM 2.5 in Changsha City, China

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          Abstract

          With the advancement of urbanization, the harm caused to human health by PM 2.5 pollution has been receiving increasing attention worldwide. In order to increase public awareness and understanding of the damage caused by PM 2.5 in the air and gain the attention of relevant management departments, Changsha City is used as the research object, and the environmental quality data and public health data of Changsha City from 2013 to 2017 are used. All-cause death, respiratory death, cardiovascular death, chronic bronchitis, and asthma were selected as the endpoints of PM 2.5 pollution health effects, according to an exposure–response coefficient, Poisson regression model, and health-impact-assessment-related methods (the Human Capital Approach, the Willingness to Pay Approach, and the Cost of Illness Approach), assessing the health loss and economic loss associated with PM 2.5. The results show that the pollution of PM 2.5 in Changsha City is serious, which has resulted in extensive health hazards and economic losses to local residents. From 2013 to 2017, when annual average PM 2.5 concentrations fell to 10 μg/m 3, the total annual losses from the five health-effect endpoints were $2788.41 million, $2123.18 million, $1657.29 million, $1402.90 million, and $1419.92 million, respectively. The proportion of Gross Domestic Product (GDP) in the current year was 2.69%, 1.87%, 1.34%, 1.04% and 0.93%, respectively. Furthermore, when the concentration of PM 2.5 in Changsha City drops to the safety threshold of 10 μg/m 3, the number of affected populations and health economic losses can far exceed the situation when it falls to 35 μg/m 3, as stipulated by the national secondary standard. From 2013 to 2017, the total loss under the former situation was 1.48 times, 1.54 times, 1.86 times, 2.25 times, and 2.33 times that of the latter, respectively. Among them, all-cause death and cardiovascular death are the main sources of health loss. Taking 2017 as an example, when the annual average concentration dropped to 10 μg/m 3, the health loss caused by deaths from all-cause death and cardiovascular disease was 49.16% of the total loss and 35.73%, respectively. Additionally, deaths as a result of respiratory disease, asthma, and chronic bronchitis contributed to 7.31%, 7.29%, and 0.51% of the total loss, respectively. The research results can provide a reference for the formulation of air pollution control policies based on health effects, which is of great significance for controlling air pollution and protecting people’s health.

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          Fine-particulate air pollution and life expectancy in the United States.

          Exposure to fine-particulate air pollution has been associated with increased morbidity and mortality, suggesting that sustained reductions in pollution exposure should result in improved life expectancy. This study directly evaluated the changes in life expectancy associated with differential changes in fine particulate air pollution that occurred in the United States during the 1980s and 1990s. We compiled data on life expectancy, socioeconomic status, and demographic characteristics for 211 county units in the 51 U.S. metropolitan areas with matching data on fine-particulate air pollution for the late 1970s and early 1980s and the late 1990s and early 2000s. Regression models were used to estimate the association between reductions in pollution and changes in life expectancy, with adjustment for changes in socioeconomic and demographic variables and in proxy indicators for the prevalence of cigarette smoking. A decrease of 10 microg per cubic meter in the concentration of fine particulate matter was associated with an estimated increase in mean (+/-SE) life expectancy of 0.61+/-0.20 year (P=0.004). The estimated effect of reduced exposure to pollution on life expectancy was not highly sensitive to adjustment for changes in socioeconomic, demographic, or proxy variables for the prevalence of smoking or to the restriction of observations to relatively large counties. Reductions in air pollution accounted for as much as 15% of the overall increase in life expectancy in the study areas. A reduction in exposure to ambient fine-particulate air pollution contributed to significant and measurable improvements in life expectancy in the United States. 2009 Massachusetts Medical Society
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            Air pollution and lung cancer incidence in 17 European cohorts: prospective analyses from the European Study of Cohorts for Air Pollution Effects (ESCAPE).

            Ambient air pollution is suspected to cause lung cancer. We aimed to assess the association between long-term exposure to ambient air pollution and lung cancer incidence in European populations. This prospective analysis of data obtained by the European Study of Cohorts for Air Pollution Effects used data from 17 cohort studies based in nine European countries. Baseline addresses were geocoded and we assessed air pollution by land-use regression models for particulate matter (PM) with diameter of less than 10 μm (PM10), less than 2·5 μm (PM2·5), and between 2·5 and 10 μm (PMcoarse), soot (PM2·5absorbance), nitrogen oxides, and two traffic indicators. We used Cox regression models with adjustment for potential confounders for cohort-specific analyses and random effects models for meta-analyses. The 312 944 cohort members contributed 4 013 131 person-years at risk. During follow-up (mean 12·8 years), 2095 incident lung cancer cases were diagnosed. The meta-analyses showed a statistically significant association between risk for lung cancer and PM10 (hazard ratio [HR] 1·22 [95% CI 1·03-1·45] per 10 μg/m(3)). For PM2·5 the HR was 1·18 (0·96-1·46) per 5 μg/m(3). The same increments of PM10 and PM2·5 were associated with HRs for adenocarcinomas of the lung of 1·51 (1·10-2·08) and 1·55 (1·05-2·29), respectively. An increase in road traffic of 4000 vehicle-km per day within 100 m of the residence was associated with an HR for lung cancer of 1·09 (0·99-1·21). The results showed no association between lung cancer and nitrogen oxides concentration (HR 1·01 [0·95-1·07] per 20 μg/m(3)) or traffic intensity on the nearest street (HR 1·00 [0·97-1·04] per 5000 vehicles per day). Particulate matter air pollution contributes to lung cancer incidence in Europe. European Community's Seventh Framework Programme. Copyright © 2013 Elsevier Ltd. All rights reserved.
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              Effects of long-term exposure to air pollution on natural-cause mortality: an analysis of 22 European cohorts within the multicentre ESCAPE project

              Few studies on long-term exposure to air pollution and mortality have been reported from Europe. Within the multicentre European Study of Cohorts for Air Pollution Effects (ESCAPE), we aimed to investigate the association between natural-cause mortality and long-term exposure to several air pollutants. We used data from 22 European cohort studies, which created a total study population of 367,251 participants. All cohorts were general population samples, although some were restricted to one sex only. With a strictly standardised protocol, we assessed residential exposure to air pollutants as annual average concentrations of particulate matter (PM) with diameters of less than 2.5 μm (PM2.5), less than 10 μm (PM10), and between 10 μm and 2.5 μm (PMcoarse), PM2.5 absorbance, and annual average concentrations of nitrogen oxides (NO2 and NOx), with land use regression models. We also investigated two traffic intensity variables-traffic intensity on the nearest road (vehicles per day) and total traffic load on all major roads within a 100 m buffer. We did cohort-specific statistical analyses using confounder models with increasing adjustment for confounder variables, and Cox proportional hazards models with a common protocol. We obtained pooled effect estimates through a random-effects meta-analysis. The total study population consisted of 367,251 participants who contributed 5,118,039 person-years at risk (average follow-up 13.9 years), of whom 29,076 died from a natural cause during follow-up. A significantly increased hazard ratio (HR) for PM2.5 of 1.07 (95% CI 1.02-1.13) per 5 μg/m(3) was recorded. No heterogeneity was noted between individual cohort effect estimates (I(2) p value=0.95). HRs for PM2.5 remained significantly raised even when we included only participants exposed to pollutant concentrations lower than the European annual mean limit value of 25 μg/m(3) (HR 1.06, 95% CI 1.00-1.12) or below 20 μg/m(3) (1.07, 1.01-1.13). Long-term exposure to fine particulate air pollution was associated with natural-cause mortality, even within concentration ranges well below the present European annual mean limit value. European Community's Seventh Framework Program (FP7/2007-2011). Copyright © 2014 Elsevier Ltd. All rights reserved.
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                Author and article information

                Journal
                Int J Environ Res Public Health
                Int J Environ Res Public Health
                ijerph
                International Journal of Environmental Research and Public Health
                MDPI
                1661-7827
                1660-4601
                11 June 2019
                June 2019
                : 16
                : 11
                : 2063
                Affiliations
                [1 ]The School of Resource, Environment and Safety Engineering, Hunan University of Science and Technology, Xiangtan 411201, China; wff390@ 123456163.com (F.W.); hujing081@ 123456163.com (J.H.); 15197292263@ 123456163.com (X.L.)
                [2 ]South China Institute of Environmental Science, Ministry of Ecology and Environment (MEE), Guangzhou 510655, China; liaoyan@ 123456scies.org
                Author notes
                [* ]Correspondence: ygh@ 123456hnust.edu.cn ; Tel.: +86-731-5829-0040 or +86-137-6220-0628
                Author information
                https://orcid.org/0000-0001-9204-5422
                https://orcid.org/0000-0001-9881-7729
                Article
                ijerph-16-02063
                10.3390/ijerph16112063
                6604026
                31212685
                f8359f64-4ee2-46a0-a2d0-2603011868ca
                © 2019 by the authors.

                Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license ( http://creativecommons.org/licenses/by/4.0/).

                History
                : 18 April 2019
                : 06 June 2019
                Categories
                Article

                Public health
                pm2.5,exposure–response coefficient,health economic loss,changsha
                Public health
                pm2.5, exposure–response coefficient, health economic loss, changsha

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