Upregulation of microRNA-424 relieved diabetic nephropathy by targeting Rictor through mTOR Complex2/Protein Kinase B signaling.

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Abstract

To investigate the role of miR-424 in diabetic nephropathy (DN) and its relationship with Rictor in mammalian target of rapamycin (mTOR) C2/Akt signaling.

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Journal

Journal ID (iso-abbrev): J Cell Physiol

Title: Journal of cellular physiology

Publisher: Wiley

ISSN (Electronic): 1097-4652

ISSN (Print): 0021-9541

Publication date (Electronic): July 2019

Volume: 234

Issue: 7

Affiliations

[1 ] Department of endocrinology, The Affiliated Lianyungang Hospital of Xuzhou Medical University, The First People's Hospital of Lianyungang, The Affiliated Hospital of Kangda College of Nanjing Medical University Lianyungang Clinical College of Nanjing Medical University, Lianyungang, China.

Article

DOI: 10.1002/jcp.27822

PubMed ID: 30637733

SO-VID: 7e40d3c2-49a0-479f-ac49-4cb89a671b9c

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Keywords: Akt,microRNA-424 (miRNA-424),mammalian target of rapamycin (mTOR),diabetic nephropathy (DN),Rictor

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Keywords: Akt, microRNA-424 (miRNA-424), mammalian target of rapamycin (mTOR), diabetic nephropathy (DN), Rictor

Upregulation of microRNA-424 relieved diabetic nephropathy by targeting Rictor through mTOR Complex2/Protein Kinase B signaling.

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Neuronal Signaling

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