ApoE-deficient mice are a model of lipoprotein oxidation in atherogenesis. Demonstration of oxidation-specific epitopes in lesions and high titers of autoantibodies to malondialdehyde-lysine in serum

Plump, A S; Witztum, J L; Steinberg, D.; Palinski, W; Ord, V A; Breslow, J L

doi:10.1161/01.ATV.14.4.605

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ApoE-deficient mice are a model of lipoprotein oxidation in atherogenesis. Demonstration of oxidation-specific epitopes in lesions and high titers of autoantibodies to malondialdehyde-lysine in serum

Author(s): W. Palinski , V. A. Ord , A. S. Plump , J. L. Breslow , D. Steinberg , J. L. Witztum

Publication date Created: April 01 1994

Journal: Arteriosclerosis, Thrombosis, and Vascular Biology

Publisher: Ovid Technologies (Wolters Kluwer Health)

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Title: Arteriosclerosis, Thrombosis, and Vascular Biology

Abbreviated Title: Arteriosclerosis, Thrombosis, and Vascular Biology

Publisher: Ovid Technologies (Wolters Kluwer Health)

ISSN (Print): 1079-5642

ISSN (Electronic): 1524-4636

Publication date Created: April 01 1994

Publication date (Print): April 01 1994

Volume: 14

Issue: 4

Pages: 605-616

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DOI: 10.1161/01.ATV.14.4.605

SO-VID: 0fc74ca5-ea80-401a-b7bf-a72cfd29ac16

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ApoE-deficient mice are a model of lipoprotein oxidation in atherogenesis. Demonstration of oxidation-specific epitopes in lesions and high titers of autoantibodies to malondialdehyde-lysine in serum

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