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      Anti-allergic Effect of Intranasal Administration of Type-A Procyanidin Polyphenols Based Standardized Extract of Cinnamon Bark in Ovalbumin Sensitized BALB/c Mice : ANTI-ALLERGIC EFFECTS OF STANDARDIZED CINNAMON BARK EXTRACT

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      Journal: Phytotherapy Research
      Publisher: Wiley-Blackwell

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          Isolation and characterization of polyphenol type-A polymers from cinnamon with insulin-like biological activity.

          Jesse M. Polansky, C Broadhurst, Norberta W Schoene … (2004)
          The causes and control of type 2 diabetes mellitus are not clear, but there is strong evidence that dietary factors are involved in its regulation and prevention. We have shown that extracts from cinnamon enhance the activity of insulin. The objective of this study was to isolate and characterize insulin-enhancing complexes from cinnamon that may be involved in the alleviation or possible prevention and control of glucose intolerance and diabetes. Water-soluble polyphenol polymers from cinnamon that increase insulin-dependent in vitro glucose metabolism roughly 20-fold and display antioxidant activity were isolated and characterized by nuclear magnetic resonance and mass spectroscopy. The polymers were composed of monomeric units with a molecular mass of 288. Two trimers with a molecular mass of 864 and a tetramer with a mass of 1152 were isolated. Their protonated molecular masses indicated that they are A type doubly linked procyanidin oligomers of the catechins and/or epicatechins. These polyphenolic polymers found in cinnamon may function as antioxidants, potentiate insulin action, and may be beneficial in the control of glucose intolerance and diabetes.
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            The united allergic airway: Connections between allergic rhinitis, asthma, and chronic sinusitis

            Charles H. Feng, Michaela D. Miller, Ronald A. Simon (2012)
            Background: The united allergic airway is a theory that connects allergic rhinitis (AR), chronic rhinosinusitis, and asthma, in which seemingly disparate diseases, instead of being thought of separately, are instead viewed as arising from a common atopic entity. Objective: This article describes patients with such diseases; explores ideas suggesting a unified pathogenesis; elucidates the various treatment modalities available, emphasizing nasal corticosteroids and antihistamines; and provides an update of the literature. Methods: A literature review was conducted. Conclusion: The aggregation of research suggests that AR, asthma, and chronic rhinosinusitis are linked by the united allergic airway, a notion that encompasses commonalities in pathophysiology, epidemiology, and treatment.
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              The role of human mast cell-derived cytokines in eosinophil biology.

              David Chi, Bita Shakoory, Robert S. Fitzgerald … (2004)
              Eosinophil-mediated diseases, such as allergic asthma, eosinophilic fasciitis, and certain hypersensitivity pulmonary disorders, are characterized by eosinophil infiltration and tissue injury. Mast cells and T cells often colocalize to these areas. Recent data suggest that mast cells can contribute to eosinophil-mediated inflammatory responses. Activation of mast cells can occur by antigen and immunoglobulin E (IgE) via the high-affinity receptor (FcepsilonRI) for IgE. The liberation of proteases, leukotrienes, lipid mediators, and histamine can contribute to tissue inflammation and allow recruitment of eosinophils to tissue. In addition, the synthesis and expression of a plethora of cytokines and chemokines (such as granulocyte-macrophage colony-stimulating factor [GM-CSF], interleukin-1 [IL-1], IL-3, IL-5, tumor necrosis factor-alpha [TNF-alpha], and the chemokines IL-8, regulated upon activation normal T cell expressed and secreted [RANTES], monocyte chemotactic protein-1 [MCP-1], and eotaxin) by mast cells can influence eosinophil biology. Stem cell factor (SCF)-c-kit, cytokine-cytokine receptor, and chemokine-chemokine receptor (CCR3) interactions leading to nuclear factor kappaB (NF-kappaB), mitogen-activated protein kinase (MAPK) expression, and other signaling pathways can modulate eosinophil function. Eosinophil hematopoiesis, activation, survival, and elaboration of mediators can all be regulated thus by mast cells in tissue. Moreover, because eosinophils can secrete SCF, eosinophils can regulate mast cell function in a paracrine manner. This two-way interaction between eosinophils and mast cells can pave the way for chronic inflammatory responses in a variety of human diseases. This review summarizes this pivotal interaction between human mast cells and eosinophils.
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                Author and article information

                Journal
                Title: Phytotherapy Research
                Abbreviated Title: Phytother. Res.
                Publisher: Wiley-Blackwell
                ISSN: 0951418X
                Publication date Created: March 2015
                Publication date (Print): March 2015
                Volume: 29
                Issue: 3
                Pages: 423-433
                Article
                DOI: 10.1002/ptr.5269
                PubMed ID: 25504814
                SO-VID: 1d3a2f5d-8691-47fd-8655-8aee88ccf0df
                Copyright © © 2015
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                http://doi.wiley.com/10.1002/tdm_license_1.1

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