Upregulation of microRNA‐424 relieved diabetic nephropathy by targeting Rictor through
      mTOR Complex2/Protein Kinase B signaling

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Abstract

To investigate the role of miR-424 in diabetic nephropathy (DN) and its relationship with Rictor in mammalian target of rapamycin (mTOR) C2/Akt signaling.

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Contributors

Guofeng Wang: (View ORCID Profile)

Journal

Title: Journal of Cellular Physiology

Abbreviated Title: J Cell Physiol

Publisher: Wiley

ISSN (Print): 0021-9541

ISSN (Electronic): 1097-4652

Publication date Created: December 07 2018

Publication date Created: July 2019

Publication date (Electronic): January 13 2019

Publication date (Print): July 2019

Volume: 234

Issue: 7

Pages: 11646-11653

Affiliations

[1 ]Department of endocrinologyThe Affiliated Lianyungang Hospital of Xuzhou Medical University, The First People's Hospital of Lianyungang, The Affiliated Hospital of Kangda College of Nanjing Medical University Lianyungang Clinical College of Nanjing Medical UniversityLianyungang China

Article

DOI: 10.1002/jcp.27822

PubMed ID: 30637733

SO-VID: 7e40d3c2-49a0-479f-ac49-4cb89a671b9c

License:

http://onlinelibrary.wiley.com/termsAndConditions#vor

http://doi.wiley.com/10.1002/tdm_license_1.1

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Upregulation of microRNA‐424 relieved diabetic nephropathy by targeting Rictor through mTOR Complex2/Protein Kinase B signaling

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